这表明在水稻基因组中广泛存在豆科植物结瘤素基因的同源基因。
This result indicated that leguminous nodulin homologous genes existed extensively in rice genome.
推测在水稻中可能存在结瘤因子所诱导的豆科早期结瘤素表达的类似机制。
Moreover, GUS gene expression driven by the Gmenod2B promoter in transgenic rice was regulated by nitrogen status.
推测可能是由于水稻中缺少了这些豆科结瘤素基因，导致水稻不能结瘤固氮。
It was suggested that the lack of these leguminous nodulin genes in rice might result in its disablility for nodulation and nitrogen fixation.
推测可能是由于水稻中缺少了这些豆科结瘤素基因，导致水稻不能结瘤固氮。
It was suggested that the lack of these leguminous nodulin genes in rice might result in its disablility for nodulation and nitrogen fixation.
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