所有这三种受影响的基因通过编码酶调节溶解体，溶解体是细胞废物处理单元。
All three affected genes encode enzymes that regulate lysosomes, the cell's waste-disposal units.
据1月12日的《细胞》杂志上的一篇研究报告，有可能通过修补一种可调节pten活性的酶来加强其基因的表达。
It might be possible to superpower the gene, known as PTEN, by tinkering with an enzyme that regulates its activity, scientists report in the Jan. 12 issue of Cell.
体外实验中对相应受体进行刺激后导致对细胞外信号调节酶（ERK）的激活有促进作用。
Stimulation of this receptor in vitro results in up-regulation of activated extracellular signal–regulated kinase (ERK).
泛素蛋白酶体途径是调节多种细胞生物学过程的重要机制，也是恶性肿瘤相关调节异常的潜在靶点；
Ubiquitin-proteasome pathway is an important mechanism regulating many processes of cellular biology, and also a potential target for abnormal regulation associated with malignancy.
泛素酶usp17在肿瘤活检标本中高表达，受细胞周期调节，且是细胞由G 1期进入S期的必需者。
The Deubiquitinating Enzyme USP17 Is Highly Expressed in Tumor Biopsies, Is Cell Cycle Regulated, and Is Required for G1-S Progression.
从而，调制了许多细胞的重要功能，包括DNA的合成及修复、蛋白质的合成与酶的活化及调节。
This, in turn, regulates many of the cell's vital functions, such as the synthesis and repair of DNA, the synthesis of proteins and the activation and regulation of enzymes.
第10号染色体同源丢失性磷酸酶-张力蛋白基因(PTEN)作为肿瘤抑制基因，可以负性调节肿瘤细胞的生长。
As a tumor-suppressor gene, phosphatase and tensin homology deleted on chromosome 10 (PTEN) regulates negatively the growth of tumor cells.
另外，表面半乳糖基转移酶通过调节表皮生长因子受体信号传导能力向胞内传递生长抑制信号，在细胞增殖控制中起重要作用。
Surface GalTase also delivers a growth inhibitory signal by modulating the ability of the EGF receptor to transduce EGF dependent signals, and plays an important role during cell growth.
细胞外基质是构成血管壁的主要成分，其代谢平衡受基质金属蛋白酶的调节。
Extracellular matrix is the main composition which constitute blood vessel wall, its balance of metabolism is to suffer the matrix metalloproteinases to regulate.
细胞膜结构的保持和细胞内区域化结构的完整有助于保持果实的采后品质，磷脂酶D活性的调节在其中起重要作用；
Preservation of membrane structure and compartmentalization helps maintain the quality of the fruits, in which the regulation of phospholipase D (PLD) plays an important role.
端粒酶的主要功能是复制端粒末端dna，维持端粒长度。端粒酶活性调节与肿瘤发生和细胞衰老有着密切关系。
The main function of telomerase is for the replication of telomere DNA, which is closely related with tumor and cell senescence.
结果抵当汤改良方能有效减轻主动脉脂质斑块面积，调节脂代谢紊乱，提高机体抗氧化酶活性，降低主动脉脂质斑块部神经酰胺含量及泡沫细胞凋亡数量。
RESULTS IPDT reduced lipid plaque area of aortic endothelium, regulated lipid metabolism, improved antioxidation of organism, ced ceramide content of aorta lipid plaque and apoptosis of foam cell.
结论：巨噬细胞内脂质过氧化物水平与酸性磷酸酶活力呈负相关，且可被免疫抑制剂和免疫激活剂所调节。
CONCLUSION: the level of LPO was negatively related to the activity of ACP, and it was regulated by immunomodulators.
机体对脂肪细胞增殖和肥大的调节机制非常复杂，涉及生长因子、激素、转录因子、脂肪代谢相关酶等。
The regulatory mechanism about hyperplasia and hypertrophy of adipose cell is very complicated. The regulators about fat metabolism include growth factors, hormones, transcription factors and enzymes.
当细胞被裂解提取全细胞提取物时，会发生正常的细胞调节缺失，并且细胞提取物中的内源性蛋白酶可以在失控的情况下使蛋白发生降解。
A loss of normal cellular control occurs during cell lysis, and endogenous proteases within the cell extract are free to degrade proteins in an uncontrolled manner.
前言： 目的：探讨端粒酶的调节基因TRAP在皮肤基底细胞癌中的表达情况。
Objectives:To investigate the expression of telomerase regulation gene TRAP in the basal cell carcinoma.
下面就细胞外基质金属蛋白酶诱导因子的作用机制、调节以及与口腔颌面部肿瘤的关系作一综述。
The advanced research about action mechanism and regulation of EMMPRIN, as well as relationship between EMMPRIN and oral and maxillofacial tumors, will be reviewed in this article.
这些观察提示突变的酪氨酸磷酸酶是肿瘤抑制基因，它调节的细胞通路可能会被利用到治疗干预中去。
These observations suggest that the mutated tyrosine phosphatases are tumor suppressor genes, regulating cellular pathways that may be amenable to therapeutic intervention.
下面就细胞外基质金属蛋白酶诱导因子的作用机制、调节以及与口腔颌面部肿瘤的关系作一综述。
The advanced research about action mechanism and regulation of EMMPRIN, as well as relationship between EMMPRIN and oral and maxillofacial tumors, will be rev…
蛋白是一个调节gtp水解酶的超家族，在细胞增殖和分化信号转导过程中发挥关键性作用。
G protein is a superfamily of regulatory GTP hydrolases. They play pivotal roles in the signal transduction of cell growth and differentiation.
泛素- 蛋白酶体途径介导的蛋白降解是机体调节细胞内蛋白水平与功能的一个重要机制。
Protein degradation mediated by ubiquitin-proteasome pathway is an important mechanism which modulates cellular proteins′ activity and function.
Chk1发挥其细胞周期检验点调节机制部分经由调控磷酸酶家族的cdc25。
Chk1 exerts its checkpoint mechanism on the cell cycle, in part, by regulating the cdc25 family of phosphatases.
周期素d 1编码一种全酶的调节亚单位，该酶利用蛋白磷酸化灭活视网膜母细胞瘤蛋白，通过细胞周期G 1 - S期促进肿瘤进展恶化。
Cyclin D1 encodes the regulatory subunit of a holoenzyme that phosphorylates and inactivates the retinoblastoma protein and promotes progression through the G1-S phase of the cell cycle.
蛋白酶体是细胞的蛋白质降解机制，由一个核心颗粒和两个调节颗粒组成。
The proteasome is essential for the selecive degradation of most cellular proteins, but how cells maintain adequate amounts of proteasome is unclear.
蛋白酶体是细胞的蛋白质降解机制，由一个核心颗粒和两个调节颗粒组成。
The proteasome is essential for the selecive degradation of most cellular proteins, but how cells maintain adequate amounts of proteasome is unclear.
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