采用不同培养时间的根霉全细胞脂肪酶催化合成己酸乙酯。
Rhizopus chinensis whole-cell lipases of different culture time were used to catalyzesynthesis of ethyl hexanoate.
治疗组肝细胞脂肪变性程度无显著变化。
And the degree of hepatocyte steatosis did not change in the treatment group.
我们假定活性氧簇分子参与了脂多糖诱导的肝细胞脂肪堆积。
We hypothesize that reactive oxygen species (ROS) mediate lipopolysaccharide (LPS) -evoked hepatic lipid accumulation.
模型组大鼠肝小叶结构紊乱，有大量纤维组织沉积，炎症细胞浸润，肝细胞脂肪变性。
Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue inflammatory cell infiltration and fatty degeneration of hepatocytes.
结论中药脂肝宁通过抑制氧化应激、稳定肝细胞膜、抑制脂质过氧化来改善酒精所致肝细胞脂肪变性。
The steatosis state of liver was improved. Conclusion ZGN improves alcoholic fatty liver by suppressing oxidative stress, stabilizing the hepatic cellular membrane and inhabiting lipid peroxidation.
事实上，白细胞介素- 1在这种情况下可以诱导肝细胞脂肪沉积，这是非酒精性脂肪性肝病的一个特征。
In fact, interleukin-1 in this scenario can induce fat deposition in the hepatocytes which is one of the characteristics on non-alcoholic fatty liver disease.
脂肪性肝病是多种原因引起的病变主体在肝小叶、以肝细胞脂肪变性和脂肪蓄积过多为主的临床病理综合征。
Fatty liver disease (FLD) is a clinicopathological syndrome caused by a variety of reasons. The main lesions are in hepatic lobule with steatohepatitis and accumulation of triglycerides in the liver.
研究人员发现，对脂肪细胞深层的影响可能是最显著的。
It was the effects deep within the fat cells that may have been the most significant, the researchers found.
离心机将脂肪细胞从干细胞和再生细胞中分离出来，将它们浓缩成一个小球，再萃取出来。
A centrifuge separates the fat cells from the stem and regenerative cells, concentrating them into a pellet, which is then extracted.
脂肪组织来源于成脂肪细胞。
脱脂奶不但只有一半的热量，它事实上还含有更多的钙质和磷，这些都可以增加细胞中脂肪的分解。
Not only does skimmed milk have half the calories, it actually has more calcium and phosophorous, which increase fat breakdown in cells.
不过，与激光疗法(利用生化途径在脂肪细胞中形成小孔)不同，它们能产生机械力量分解这些细胞。
But unlike the laser treatments, which exploit biochemical pathways to create pores in the fat cell, they generate mechanical forces that rupture these cells.
现在，脂肪细胞不能复制自己。
此外，脑组织内含有许多可氧化的物质，特别是绕在神经细胞周围的脂肪膜。
Moreover, brain tissue contains a great deal of oxidisable material, particularly in the fatty membranes surrounding nerve cells.
所以结果是一样的——越来越多的脂肪细胞。
主要的“罪犯”是低密度脂蛋白胆固醇，它是身体的运油车，在血液里向细胞传输脂肪和胆固醇。
The main culprit, LDL (for low-density lipoprotein), is the body's oil truck, circulating in the blood, delivering fat and cholesterol to the cells.
研究的目的是要看这些植物多酚是否能够对抗动物如人的脂肪细胞。
The idea is to see if these plant polyphenols could be translated into fighting fat cells in animals, i.e. humans.
最终结果是，会产生更大更多的脂肪细胞。
Ultimately, this leads to larger fat cells, and more of them.
脂肪细胞为了告诉你已经吃饱了，会产生一种激素——瘦素。
Leptin is a hormone made by fat cells that tells you when you're full.
那么，那些脂肪漏出细胞后，发生了什么变化？
Sowhat happens to the fat once it has leaked out of the cell?
该病毒还会增加新脂肪细胞的脂肪灵敏度，并降低其瘦素分泌。
The virus also increases lipid sensitivity and decreases leptin secretion of the new fat cells.
已知植物多酚阻止脂肪细胞的发育。
Plant polyphenols are known to resist the development of fat cells.
该病毒会感染脂肪组织，然后复制、分化、积累脂肪细胞。
The virus infects the fatty tissue and increases replication, differentiation, and accumulation of fat cells.
消瘦素消除你的食欲，它来自脂肪细胞。
膳食中过量的卡路里储存于白色脂肪细胞中，囤积于身体，特别是大腿和腹部。
Excess caloric energy in the diet is stored in white fat calls that pile up in the body, particularly in the thighs and abdomen.
不沾一切动物产品的人，显然根本没有动物脂肪，他们在肌肉细胞内的脂肪积累似大为减少，他们患糖尿病的几率极低。
People who avoid all animal products obviously get no animal fat at all, they appear to have much less fat build-up inside their cells, and their risk of diabetes is extremely low.
这些干细胞再和剩余的脂肪组织细胞混合。
Those cells are then combined with some of the remaining liposuctioned fat-tissue cells.
Leptin由脂肪细胞分泌，能够抑制食欲同时增强新陈代谢的水平。
Leptin, a hormone produced by fat cells, decreases appetite and boosts metabolism: low leptin levels can lead to an increased appetite.
Leptin由脂肪细胞分泌，能够抑制食欲同时增强新陈代谢的水平。
Leptin, a hormone produced by fat cells, decreases appetite and boosts metabolism: low leptin levels can lead to an increased appetite.
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