单独使用三种药都要抑制APL和ALL细胞株的生长但不会直接导致细胞死亡，而联合使用可以引起表型分化。
ALL three drugs inhibited the clonogenic growth of the APL and ALL cell lines without inducing immediate apoptosis, but associated with induction of phenotypic differentiation.
结论AT P不仅抑制人胃癌细胞的增殖，而且诱导分化，使人胃癌细胞的恶性表型向正常表型逆转。
Conclusion ATP not only inhibits proliferation but also induces differentiation of a human gastric cancer cell line with reversion from malignant toward normal phenotype.
牙周韧带细胞的表型分化及其表面标记物。
Phenotype differentiation and surface marker of periodontal ligament cell.
对扩增的细胞进行免疫表型、定向分化、造血祖细胞集落分析及脾集落形成单位的研究。
The expanded cells were further analyzed by phenotype, committed differentiation, progenitor colony assay as well as colony forming unite spleen.
目的探讨骨髓基质细胞分化为成骨表型的诱导条件。
ObjectiveTo explore environmental conditions under which bone marrow stromal cells could be induced into osteogenic phenotype.
结论人肾上腺微血管内皮细胞是特殊分化的，具有不同于其他器官组织内皮细胞的表型和功能特性。
Conclusion Human AdrEC are specially differentiated and have characteristics that are different from other organ-derived MVEC in phenotypes and functions.
分离的细胞通过特定培养基培养三天诱导分化为表达胰脏内分泌物表型的细胞。
The cells were induced to differentiate into a pancreatic endocrine phenotype by defined culture conditions within 3 days.
结果表明，突变型P53蛋白的表达与脑肿瘤的组织类型，分化程度及细胞的增殖有关，而它的高表达又可能是肿瘤恶性表型或转移的标志之一。
This results showed that mutant P53 expression was associated with a high Ki-67 LI and high histologic grade of tumors. P53 overexpression might be a genetic marker of tumor malignancy.
结果表明，突变型P53蛋白的表达与脑肿瘤的组织类型，分化程度及细胞的增殖有关，而它的高表达又可能是肿瘤恶性表型或转移的标志之一。
This results showed that mutant P53 expression was associated with a high Ki-67 LI and high histologic grade of tumors. P53 overexpression might be a genetic marker of tumor malignancy.
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