这些栓子一般由脂肪、胆固醇、钙和炎性细胞组成。
These blockages are made of fat, cholesterol, calcium, and inflammatory cells.
有栓子脱落患者的颈动脉粥样硬化斑块具有更明显的炎性细胞浸润。
The carotid atherosclerotic plaques in patients with embolus detachment had more obvious inflammatory cellular infiltration.
血流变学改变、红细胞串珠样改变、红细胞膜变形性减低、血小板聚集率增高，血浆粘度及流动栓子增多是脑梗死发病的重要病理基础。
The change of membrane mobility, the raising of platelet assemble rate, the in creasing of plasma viscosity and flowing embolismare the important pathological basis of cerebral infarction .
血流变学改变、红细胞串珠样改变、红细胞膜变形性减低、血小板聚集率增高，血浆粘度及流动栓子增多是脑梗死发病的重要病理基础。
The change of membrane mobility, the raising of platelet assemble rate, the in creasing of plasma viscosity and flowing embolismare the important pathological basis of cerebral infarction .
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