白细胞介素-12（IL-12）最初称作自然杀伤细胞刺激因子（NKSF）或细胞毒性淋巴细胞成熟因子（CLMF），是一种异二聚体细胞因子。
Interleukin-12 (IL-12) previously called cytotoxic lymphocyte maturation factor (CLMF) or NK cell stimulatory factor (NKSF) is a recently characterized heterodimeric cytokine composed of two subunits.
研究表明，组蛋白的乙酰化与去乙酰化状态，可作用于T、B细胞的发育、分化、成熟、分泌及表面因子的表达等多个方面。
Studies had shown that acetylation and deacetylation of histone are of certain value on t, B cell's development, differentiation, maturation, secretion and surface factor's expression etc.
目的：探讨造血刺激因子对巨核系成熟细胞体外扩增的影响。
Objective To investigate the effect of hematopoietic stimulating factors on the expansion of mature megakaryocytes.
免疫活性肽(ICP)和表皮生长因子(EGF)是两种生物活性因子，ICP对牛卵母细胞体外成熟及胚胎体外培养的影响还未见报道。
Immunocompentent peptide (ICP) and epidermal growth factor (EGF) are two bio - active factors, the effect of ICP on in vitro maturation of bovine oocytes and development of embryos not being reported.
这和最近的研究是一致的，该研究表明卵母细胞成熟抑制因子能诱导用TNF处理的人类神经多肽的凋亡而不从线粒体中释放出来（7）。
This is in accord with a recent study that shows Omi can induce apoptosis in human neutrophils treated with TNF- without being released from the mitochondria (7).
以中风的状况来说，细胞会死去或无法成熟，因此找出哪些生长因子可以支持神经生存，以及教导未成熟细胞变成健康而富连结的神经细胞，就很重要了。
In stroke, where cells die or fail to mature, it will be important to identify growth factors that support neuronal survival and teach immature cells to become healthy, well-connected neurons.
该试验清楚地表明了卵母细胞成熟抑制因子单独决定HAX - 1的酶切，在本实验使用的条件下对于细胞凋亡是必须的。
This experiment clearly shows that Omi is solely responsible for HAX-1 cleavage, which is essential for apoptosis under the conditions used in these experiments.
一旦由包括VEGF在内的促成熟细胞因子活化后，破骨细胞的前体细胞可以分化成为成熟的破骨细胞，破坏骨组织，给肿瘤细胞的生长腾出空间。
When activated by maturation factors including VEGF, the pre-osteoclasts differentiated into mature osteoclasts and chew up bone tissue, providing the tumor new space to grow.
这暗示着卵母细胞成熟抑制因子靠酶切hax -1蛋白来启动线粒体中的细胞凋亡。
This suggests that Omi can initiate apoptosis in the mitochondria by cleaving HAX-1 protein.
这暗示着卵母细胞成熟抑制因子靠酶切hax -1蛋白来启动线粒体中的细胞凋亡。
This suggests that Omi can initiate apoptosis in the mitochondria by cleaving HAX-1 protein.
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