这项工作显示了氧化低密度脂蛋白在骨桥蛋白基因表达中的作用，并且进一步解释了氧化应激在细胞因子调节中的作用。
This work demonstrates the role of OxLDL in the expression of the OPN gene and further highlights the role of oxidative stress in the regulation of this cytokine.
目的探讨各种细胞因子对T细胞生长激素(GH)基因表达的影响。
Aim to explore the effects of cytokines on expression of growth hormone (GH) gene in t lymphocytes.
紫外线作用于成纤维细胞，引起细胞因子分泌及基因表达的改变，不仅能造成真皮层的损伤，还可被用来治疗某些疾病，如局限性硬皮病。
Ultraviolet not only causes dermal damages by changing cytokine secretion and gene expression of human skin fibroblasts, but also is used to treat some diseases such as localized scleroderma.
许多转录因子、细胞内信号转导调节蛋白、炎症细胞因子和细胞凋亡相关基因的表达均发生了明显的调节变化。
The expressions of many genes encoding transcription factor, cytokines, cell signaling modulators and apoptosis associated proteins were found to have changed.
提示肿瘤细胞膜抗原及糖蛋白表达的改变可能是细胞因子基因转导影响肿瘤细胞免疫原性的重要结构基础。
The effect of various gene transduction on the expression of tumor cell MHC antigen and lectin receptors was investigated b…
摘要本试验旨在研究饲粮能氮水平对不同品种猪肉质性状以及肌纤维类型和细胞因子相关基因表达的影响。
Abstract: This study was conducted to investigate the effects of dietary energy and nitrogen levels on meat traits, muscle fiber type and cytokine related gene expression of pigs.
炎症反应中t辅助细胞的分化，细胞因子、趋化因子等基因的表达都受到表观遗传的调控。
In inflammation responses, differentiation of t helper cells and gene expression of cytokine and chemokine are all regulated by epigenetics.
研究发现自闭症患者大脑中的炎性细胞因子、趋化因子及生长因子是过表达的。而且自闭症患者的免疫基因表达和干扰信号通路也存在极大的差异。
Cytokines, Chemokines, and Growth Factors in ASD brain are found elevated, and the immune gene expression and the Interferon signaling pathway are found significantly different in the ASD.
结论：稳定表达人ox40l的乳腺癌转基因细胞在体外能有效地活化t细胞，介导其增殖、分泌细胞因子及抑制T细胞活化诱导的细胞死亡。
CONCLUSION: OX40L-MDA-MB-435 cells could activate t cells in vitro, promote t cell proliferation and cytokine secretion, and suppress t cell activation-induced cell death.
结论：稳定表达人ox40l的乳腺癌转基因细胞在体外能有效地活化t细胞，介导其增殖、分泌细胞因子及抑制T细胞活化诱导的细胞死亡。
CONCLUSION: OX40L-MDA-MB-435 cells could activate t cells in vitro, promote t cell proliferation and cytokine secretion, and suppress t cell activation-induced cell death.
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