结论不同种类的热休克蛋白依据自身表达的特点对移植后的免疫损伤作出反应，体现了细胞的自我保护机制。
Conclusions Different molecular chaperones respond to immune injury according to their own characteristics after liver transplantation, which shows the mechanism of cellular self-protection.
本文从细胞信号传导的角度，综述热休克蛋白在运动应激下免疫机能的反应和适应过程中的作用。
Here we discussed the HSP function as a signal molecule to regulatory immune cells under exercise stress.
与未孕小鼠相比，孕鼠热休克蛋白70免疫反应阳性细胞显著增多（P< 0 .0 1 ）且随妊娠日龄的增加而增加（P< 0 .0 1 ）。
HSP 70 immunoreactivity positive cells in pregnant mouse was strikingly increased compared with control mouse ( P< 0.01), and it increased with the increasing of gestation day ages. ( P< 0.01).
与未孕小鼠相比，孕鼠热休克蛋白70免疫反应阳性细胞显著增多（P< 0 .0 1 ）且随妊娠日龄的增加而增加（P< 0 .0 1 ）。
HSP 70 immunoreactivity positive cells in pregnant mouse was strikingly increased compared with control mouse ( P< 0.01), and it increased with the increasing of gestation day ages. ( P< 0.01).
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