在应答缺血性心肌损伤时，脾单核细胞能动性增加，全体离开脾，在受伤的组织聚集，参与伤口愈合。
In response to ischemic myocardial injury, splenic monocytes increase their motility, exit the spleen en masse, accumulate in injured tissue, and participate in wound healing.
巨噬细胞在宿主防御反应中起了重要的作用，是天然免疫应答的主要参与者。
Macrophages play a significant role in the host defense mechanism. And it is the key participants in the innate immune response.
结论嵌合状态与肾移植耐受存在明显相关性，克隆不应答、抑制细胞参与了耐受的形成。
Several other mechanisms may by theorized for the induction of tolerance in this renal transplant model, including clonal anergy and suppressive cells.
结论嵌合状态与肾移植耐受存在明显相关性，克隆不应答、抑制细胞参与了耐受的形成。
Several other mechanisms may by theorized for the induction of tolerance in this renal transplant model, including clonal anergy and suppressive cells.
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