我们偶然发现送入细胞内的STAT1C过量表现会造成LL2细胞异常的STAT蛋白活化，进一步研究其机制也许能发现一些新的JAK/STAT讯息传递路径的调节系统。
We incidentally found that STAT1C overexpression cause aberrant STAT activation in LL2 cells. Further studies on the underlying mechanisms may explore a novel regulatory system of JAK/STAT pathway.
我们偶然发现送入细胞内的STAT1C过量表现会造成LL2细胞异常的STAT蛋白活化，进一步研究其机制也许能发现一些新的JAK/STAT讯息传递路径的调节系统。
We incidentally found that STAT1C overexpression cause aberrant STAT activation in LL2 cells. Further studies on the underlying mechanisms may explore a novel regulatory system of JAK/STAT pathway.
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