已被抗原致敏的细胞毒性T细胞是效应细胞，在抗病毒免疫中起到特别重要的作用。
Cytotoxic t cells, after sensitization by antigen, are effector cells, especially important in antiviral immunity.
本文研究了注射含微囊藻毒素的微囊藻细胞抽提液对小鼠血液以及免疫系统的亚慢性毒性作用。
In the present study, toxic effects of Microcystis cell extracts containing microcystin-LR on the treated-mice blood were investigated.
免疫反应所产生的NO对邻近组织和能够产生NOS的细胞也有毒性作用。
No from immunoreaction is toxic for adjacent tissues and cells capable of generating NOS.
目的：探讨氟烷性肝炎免疫应答中抗体依赖细胞介导的细胞毒作用(adcc)对肝细胞的毒性作用。
Objective: to study antibody dependent cell-mediated cytotoxicity (ADCC) on hepatocytes during immune response in halothane hepatitis of guinea pigs.
目的探讨白细胞介素（IL）2与IL4对膀胱癌肿瘤浸润性淋巴细胞（TIL）体外增殖及细胞毒性免疫调控的协同作用。
Objective To investigate the synergy effects of IL 2 and IL 4 on the proliferation and cytotoxicity of tumor infiltrating lymphocytes (TIL) from murine bladder carcinoma in vitro.
因此，外来体可能具有细胞毒性效应、免疫调节、诱导凋亡和免疫耐受诱导等作用。
Exosomes, therefore, may be in possession of CTL effect, immune regulation, inducing apoptosis immune enduring and so on.
方法观察普乐林注射液对小鼠的急性毒性，对人红细胞的体外溶血作用，对豚鼠致敏性及免疫性溶血的影响。
Methods: The acute toxicity of Puerarin in mice, the haemolytic effect on human erythrocyte in vitro, hypersensitization and immunohaemolytic effect on guinea pigs were studied.
目的研究细胞趋化因子受体5（CCR5）在乙型病毒性肝炎（HB）免疫发病机制中的作用。
Objective To investigate the role of CC chemokine receptor 5(CCR5) in the immunological pathogenesis of hepatitis B(HB). ?
目的研究梅花鹿免疫细胞活性因子(S IAF)对病毒性心肌炎的治疗作用。
Objective to study the curative effect of sika deer immunocyte activity factor (SIAF) on viral myocarditis in mouse model.
结论龙泉复方制剂是一种非常有前景的治疗肿瘤的复方中药，其作用机制可能是，对肿瘤细胞直接的毒性作用和通过促进免疫反应的间接抑瘤作用。
Conclusion Longquan Fufang is a promising Chinese medicine for tumor therapy. Its mechanism could be direct toxicity for tumor cells and indirect inhibition by immune regulation.
结论龙泉复方制剂是一种非常有前景的治疗肿瘤的复方中药，其作用机制可能是，对肿瘤细胞直接的毒性作用和通过促进免疫反应的间接抑瘤作用。
Conclusion Longquan Fufang is a promising Chinese medicine for tumor therapy. Its mechanism could be direct toxicity for tumor cells and indirect inhibition by immune regulation.
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