结论凝血酶对丘脑组织具有毒性损伤作用，但这一损伤可被水蛭素逆转。
Conclusion Damage to the thalamic tissue might be caused by thrombin and reversed by hirudin.
当组织损伤时，释放出凝血致活酶，把凝血酶原变成凝血酶。
When the tissue damage, the release of thromboplastin to prothrombin into thrombin.
结论凝血酶可以引起脑出血后脑组织水肿，水蛭素可以抑制凝血酶所引起的脑水肿且水蛭素本身不对脑组织产生负面影响。
Conclusion Thrombin can lead to brain edema after cerebral hemorrhage which can be restrained by hirudin. And then, hirudin doesn't produce any negative effect on brain tissue.
结论凝血酶可以引起脑出血后脑组织水肿，水蛭素可以抑制凝血酶所引起的脑水肿，且水蛭素本身不对脑组织产生任何负面影响。
Conclusion Thrombin lead to brain edema after cerebral hemorrhage that could be restrained by hirudin, and hirudin didn't produce any negative effect on brain tissue.
目的探讨凝血酶对脑组织损害的机制，及凝血酶抑制剂水蛭素和尼莫地平对其的影响。
Objective To investigate the mechanism of brain injury caused by thrombin and the intervention effect of hirudin and nimodipine.
论述了血液成分包括凝血酶、血红蛋白、血浆蛋白、白细胞和补体等对血肿周围组织损伤的影响。
The important pathological changes after ICH include hematoma enlarging and it's pressure effect, changes of local cerebral blood flow, poison effect of coagulum and cerebral edema.
论述了血液成分包括凝血酶、血红蛋白、血浆蛋白、白细胞和补体等对血肿周围组织损伤的影响。
The important pathological changes after ICH include hematoma enlarging and it's pressure effect, changes of local cerebral blood flow, poison effect of coagulum and cerebral edema.
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