结论大花紫玉盘素可能通过线粒体通路诱导细胞凋亡。

Conclusion Uvarigrin induced apoptosis in KBv200 cells and KB cells probably through a mitochondria-dependent pathway.

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线粒体介导了死亡信号通路中自噬与凋亡的相互作用。

There is a mutual regulation between apoptosis and autophagy in death signaling process mediated by mitochondria.

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但缺陷基因却不同产生蛋白质以维持线粒体的正常功能—尤其是神经系统细胞通路的线粒体。

But a defect in the gene may disable its proteins from performing their normal functions in mitochondria - most notably the mitochondria within the nervous system's cellular pathways.

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但缺陷基因却不同产生蛋白质以维持线粒体的正常功能—尤其是神经系统细胞通路的线粒体。

But a defect in the gene may disable its proteins from performing their normal functions in mitochondria - most notably the mitochondria within the nervous system's cellular pathways.

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