目的：探讨缺氧大鼠脑线粒体能量代谢的特点。
AIM: to explore the characteristics of energy metabolism in brain mitochondria of rats exposed to acute and chronic hypoxia.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
目的：观察异丙酚对缺血再灌注损伤大鼠海马线粒体能量代谢、ATP酶活性、脂质过氧化及超微结构的影响。
AIM: to investigate the effects of propofol on the ATP content, ATPase activity, lipid peroxidation and ultrastructure in hippocampus mitochondrial in rats following global ischemia-reperfusion.
目的：观察异丙酚对缺血再灌注损伤大鼠海马线粒体能量代谢、ATP酶活性、脂质过氧化及超微结构的影响。
AIM: to investigate the effects of propofol on the ATP content, ATPase activity, lipid peroxidation and ultrastructure in hippocampus mitochondrial in rats following global ischemia-reperfusion.
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