内皮细胞功能紊乱的根本机制在于内皮细胞线粒体活性氧簇(ROS)生成过多。
An underlying mechanism for endothelial dysfunction is the enhanced generation of endothelial mitochondrial reactive oxygen species (ROS).
就解偶联蛋白3的结构、分布、表达调控及其介导的线粒体活性氧生成等相关问题作一综述。
The article reviewed the structure, distribution, expression regulation of UCP3, and its regulation on mitochondrial reactive oxy2 gen species production.
运动可导致鼠肝脏内的酶含量、酶活性及线粒体功能发生变化。
Enzyme content and active and mitochondria function will change about in rat liver in different exercise.
目的：探讨全反式维甲酸（tRA ）对大鼠棕色脂肪组织线粒体上解偶联蛋白（UCP）活性和含量的影响。
Objective: To evaluate effects of tRA on activity and content of UCP in rat brown adipose tissue mitochondria.
心脏骤停后使用亚硝酸盐治疗能够减少活性氧生成、改善心脏和神经功能并且通过对线粒体复合物i的可逆性阻滞提高生存率。
Nitrite Therapy After Cardiac Arrest Reduces Reactive Oxygen Species Generation, Improves Cardiac and Neurological Function, and Enhances Survival via Reversible Inhibition of Mitochondrial Complex I.
结论艾司洛尔混合肾上腺素对心肺复苏后大鼠心肌有一定的保护作用，升高了线粒体atp酶活性。
Conclusion Esmolol combined with adrenaline can afford some protection on myocardium after CPR and increase ATPase activity of myocardial mitochondria.
缺氧暴露可显著抑制心肌线粒体ANT活性，并随时间呈双相效应。
Acute hypoxic exposure could lead to decrease significantly of ant transport activity in mitochondria.
肝线粒体分离后以氢电极法测定其呼吸活性。
Hepatic mitochondria were isolated and mitochondrial respiratory activity was assayed in a Clark oxygen electrode.
结果与结论：线粒体在态4呼吸状态下，DCF的荧光强度随时间呈线性增加，表明活性氧以恒定速率产生。
Results and Conclusion: DCF fluorescence showed linear increase in State 4 mitochondria, which suggest ROS produced at constant rate.
发现线粒体从生物中分离出来以后具有一定活性，并能利用自身营养物生长很长一段时间。
After isolated from biological living body, mitochondria have still activity and can live for a long time by using the nutrient in themselves.
与正常对照组比较，模型组小鼠海马神经细胞线粒体超氧化物歧化酶（SOD）活性明显降低（P<0.01），丙二醛（MDA）含量显著升高（P<0.01）；
The activity of SOD decreased(P<0.01), and the contents of MDA significantly increased(P<0.01)in the model group comparing to those in normal controls.
方法用荧光分光光度计及紫外分光光度计技术检测了去卵巢模型大鼠海马及额叶皮层的SOD、COX活性、MDA含量及线粒体膜流动性的变化。
Methods We measure the activity of SOD, MDA and COX by UV-spectrophotometer and measure the fluidity of mitochondrial membrane by fluorescence spectrophotometer.
目的：观察异丙酚对缺血再灌注损伤大鼠海马线粒体能量代谢、ATP酶活性、脂质过氧化及超微结构的影响。
AIM: to investigate the effects of propofol on the ATP content, ATPase activity, lipid peroxidation and ultrastructure in hippocampus mitochondrial in rats following global ischemia-reperfusion.
目的：探讨滋补脾阴方药对老龄大鼠脑线粒体膜ATP酶活性的影响。
Objective: To study the effect of the PNPY on the brain mitochondria ATP ase activity of senile rats.
结论粉防己碱因其钙拮抗作用，在降低血压、逆转左室肥厚的同时，可使细胞膜钠泵活性、线粒体钙泵活性恢复。
CONCLUSION Tetrandrine, as a calcium channel blockers, can reverse LVH induced by RH and elevate the cardiomyocytic membrane sodium pump and mitochondrial calcium pump activities.
目的探讨亚硒酸钠诱导NB 4细胞凋亡过程中，活性氧引起的线粒体膜电位丧失及凋亡作用的相关机制。
Objective to investigate the role of reactive oxygen species (ROS) and ROS-caused mitochondrial transmembrane potential loss in sodium selenite-induced apoptosis in NB4 cells.
用含10%猪油(重量比)的饲料喂养豚鼠50天，观察对心肌线粒体脂质构成和细胞色素氧化酶活性的影响。
Effect of 10% lard diet on lipid compositions and the activity of cytochrome oxidase of cardiac mitochondria from guinea pigs was observed.
现已发现，氧化应激可以改变端粒酶活性，促使端粒酶从细胞核转位到线粒体，因而不能继续维持端粒长度，使细胞端粒缩短。
It has been demonstrated that telomerase activity would be altered upon oxidative stress and excepted from nucleolus to mitochondria as well as telomere shortening.
主要论述了植物处于低温胁迫下其线粒体膜脂与膜蛋白的变化以及在低温伤害反应中植物所产生的活性氧对抗氰呼吸的影响。
This paper deals mainly with the effect of mitochondrial membrane lipids, membraneprotins and the produced active oxygen under low temperature on the cyanide-resistant respiration.
结果发现，肝脏脂肪变性与急性严重感染有密切关系，其脂肪变性主要与感染、毒素等损害了肝细胞内线粒体及其酶的活性有关。
The results showed that liver steatosis was closely related to the serious acute infection, in which the infection or toxin might damage enzyme and thread granules in the liver cells.
目的研究-突触核蛋白(-SYN)在大鼠不同脑区线粒体的表达及对线粒体呼吸链复合体I活性的影响。
Objective to study the expression of mitochondrial -synuclein (-syn) in different rat brain regions and its effect on complex I activity of respiratory chain.
线粒体呼吸链电子漏是运动性内源活性氧生成的重要机制之一。
The mitochondrial electron leak is an important source of endogenous ROS in exercise.
结果心肌肽素可剂量依赖性地降低MDA的含量，提高SOD活性及增强受损细胞内线粒体脱氢酶活性。
RESULTS CMP could decrease dosedependently MDA content and increase SOD activity , dehydrogenase activity was enhanced dosedependently also in mitochondria.
线粒体的 -SYN有可能来源于胞质 -SYN向线粒体的转运，并调节复合体I的活性。
Mitochondrial -SYN may come from the transportation of cytoplasmic -SYN and may regulate the complex I activity of respiratory chain.
高剂量淫羊藿苷明显抑制了心肌线粒体MDA含量，增加了心肌线粒体SOD活性。
Furthermore, high dose of icariin significantly decreased the mitochondrial MDA level and increased SOD activity in diabetic rat hearts.
对与线粒体有关的过氧化物酶、超氧化物歧化酶、细胞色素氧化酶和琥珀酸脱氢酶进行了酶活性分析。
This study deals with Jin A male sterile and its maintainer line in activity of several enzymes.
利用L KB 2 2 77生物活性检测系统，测定了家鸡两个品种的心脏和肝脏线粒体的活性及其代谢热。
The metabolic thermograms and activity of mitochondria from the heart and liver of two varieties of the chicken have been determined by using a LKB2277 bioactivity monitor.
组织化学及超微结构观察表明，模型组sdh活性下降，ACP活性增高，线粒体明显肿胀、变性、脊断裂。
According to the observation of histochemistry and ultrastructure, it showed that SDH activity decreased, ACP activity increased, the mitochondrion swelled and became deformed, crest broke.
组织化学及超微结构观察表明，模型组sdh活性下降，ACP活性增高，线粒体明显肿胀、变性、脊断裂。
According to the observation of histochemistry and ultrastructure, it showed that SDH activity decreased, ACP activity increased, the mitochondrion swelled and became deformed, crest broke.
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