相反的，纤维原细胞也发出信号的刺激肿瘤的细胞生长。
The fibroblasts, in turn, send signals to the tumor cells to stimulate their growth.
FLT3L是通过t细胞和基质纤维原细胞产生(1,2)。
FLT3L is produced by t cells and stromal fibroblasts (1, 2).
在这三个例子中，纤维原细胞均产生了成熟的棕色脂肪细胞。
In all three cases, the fibroblasts produced mature brown fat cells.
最近的研究也显示，肿瘤微环境中的纤维原细胞也会自己突变。
Recent studies suggest, too, that the fibroblasts in a tumor microenvironment begin to acquire mutations of their own.
肿瘤细胞发出信号来刺激形成支架细胞的生长，即纤维原细胞。
Tumor cells send signals to stimulate the growth of the cells that form the scaffold in which the tumor cells grow, known as fibroblasts.
酸性的纤维原细胞生长因素加速在糖尿病的小老鼠治愈的皮肤的创伤。
Acidic fibroblast growth factor accelerates dermal wound healing in diabetic mice.
酸性的纤维原细胞生长因素阻止海马趾的CA1的死亡角锥体的细胞后面的局部缺血。
Acidic fibroblast growth factor prevents death of hippocampal CA1 pyramidal cells following ischemia.
当蛋白质结构层密度足够防止细胞渗透进人造血管时，研究人员会在其表面覆盖一层纤维原细胞片。
Then the researchers layered another sheet of fibroblasts on the outside of this scaffold, which is dense enough to prevent the cells from easily migrating to the inside of the engineered vessel.
通过一个胶原质支架为完整厚皮缺点的待遇在一个野兔模型酸性的纤维原细胞生长因素的调查交付了。
Investigation of acidic fibroblast growth factor delivered through a collagen scaffold for the treatment of full-thickness skin defects in a rabbit model.
2006年，日本的一个研究员宣布他用遗传学的方法重组了老鼠纤维原细胞——一中皮肤细胞——使其具有多潜能。
In 2006 a researcher in Japan announced that he'd genetically reprogrammed mouse fibroblasts-a type of skin cell-to make them pluripotent.
在这学习通过使用一个胶原质支架的酸性的纤维原细胞生长因素的使用完整厚皮缺点的反应被提高的治愈的创伤。
In this study the wound healing response of full-thickness skin defects was enhanced through the use of acidic fibroblast growth factor using a collagen scaffold.
之后将切片卷起促使细胞产生一种由结构支撑蛋白组成的混合物，即胶原及弹性蛋白。 纤维原细胞的问题在于它们会转化为平滑的肌肉细胞，最终将血管堵塞。
They then roll up the sheet and allow the cells to produce an interpenetrating mixture of structural support proteins, known as collagen and elastin.
这些分子会”通知“血液中的凝血剂，并聚集起两种在血液中循环的成分----称为血小板的细胞碎片和称为纤维蛋白原的可溶性蛋白分子。
They round up cellular fragments known as platelets, along with molecules of a soluble protein called fibrinogen, both of which circulate in the bloodstream.
电镜下见成纤维细胞胞膜基底部向HA-ECM内伸出数个突起，细胞与羊膜基质粘附相当牢固，胞外有较多胶原原纤维堆积。
The basal membrane of fusiform fibroblasts with oval nucleus and rich in chromatin reached much protuberance into matrix of HA ECM and collagen fibril cumulating outside cells.
目的探讨肠道肌原成纤维细胞瘤（IMFTS）的临床病理及免疫组织化学特点。
Objective To explore the clinical pathological and immunohistochemical features of intestinal myogenic fibroblastic tumors(IMFTS).
目的：探讨原发于胃的恶性纤维组织细胞瘤的临床病理学特征。
AIM: to study the clinical and pathological features of malignant fibrous histiocytoma in the stomach.
而超微结构改变以软骨细胞破碎的比较多见，有的软骨细胞则增生，基质胶原原纤维明显减少。
Many broken cartilage cells could be observed and matrix collagenous fibril decreases obviously, but some cartilage cells proliferated.
未经处理的同种异体肌腱组：偶见腱细胞，未见到新生的细胶原原纤维，粗胶原原纤维排列紊乱、疏松。
Fresh allograft group: No tenocyte, and few new collagen fibril could be seen, thick collagen fibril arranged at random and loose.
目的研究硬皮病成纤维细胞整合素表达对成纤维细胞合成原胶原的影响。
Objective Investigation of the effect of integrin on fibroblasts from scleroderma in the production of procollagen.
用x射线衍射法对棉纤维细胞壁的微原纤螺旋角和纤维素微晶相对取向指数在植株中的分布规律进行了研究。
The distribution of microfibril spiral angle and relative orientational index of cellulose crystallites in cotton fibre cell walls had been studied by X-ray diffraction analysis.
目的研究肺原发恶性纤维组织细胞瘤的治疗方法及其预后。
Objective Objective to study the treatment method and prognosis of primary malignant fibrous histiocytoma of the lung.
目的探讨原发于上颌窦的恶性纤维组织细胞瘤（MFH）的病理、临床表现及治疗措施。
Objective To explore the pathology, clinical manifestation and the treatment of malignant fibrous histiocytoma (MFH) initially located in maxillary sinuses.
两组于治疗前、后分别采静脉血检测血浆纤维蛋白原、红细胞压积、全血粘度和血浆粘度的变化。
Venous blood samples were taken respectively in the two groups to detect the changes of plasma fibrinogen, hematocrit, whole blood viscosity and plasma viscosity.
结果脂肪肝患者的全血高切黏度、中切黏度、低切黏度、血浆黏度、红细胞聚集指数和纤维蛋白原均高于非脂肪肝人员（P〈0.01）。
Results All the values of whole blood viscosity, plasma viscosity, red cell aggregation index and plasma fibrinogen of the patients with fatty liver were higher than those of healthy people(P0.01).
结果输注代血浆和平衡液后全血粘度、血浆粘度、HCT、红细胞聚集指数、纤维蛋白原两组均较输注前明显降低（P<0.05），但两组间比较无显著差异（P>0.05）。
Results Blood viscosity, HCT, index of RBC aggregation and fibrinogen decreased significantly(P<0.05)in each group and there was no significant difference between the two groups(P>0.05).
毛细血管内皮细胞形态结构发生改变，基膜增厚，血管周隙明显增宽，内含大量的胶原原纤维。
The structure of endothelial cells of capillaries was changed, the basal lamina became thicker and the pericapillary space became broader with a lot of collagen fibrils.
结论降低硬皮病成纤维细胞整合素的表达，可从转录水平抑制成纤维细胞合成原胶原。
Conclusion Overproduction of procollagen may be inhibited in the level of transcription by lowering the expression of integrin on fibroblasts in scleroderma.
刮伤实验中细胞迁移速度及慢速显微摄像单细胞随机迁移速度在纤维蛋白原组及对照组细胞均无明显差别（P>0.05）。
Fibrinogen had no obvious effect on VSMC migration in scrape assay and on VSMC migration at random direction in Time-lapse microcinematography (P>0.05).
降纤酶有明显降低血黏度、纤维蛋白原含量、红细胞聚集性、血小板聚集作用。
Defibrase can effectively reduce blood viscosity, content of fibrinogen, hemocyte aggregation and platelet aggregation.
降纤酶有明显降低血黏度、纤维蛋白原含量、红细胞聚集性、血小板聚集作用。
Defibrase can effectively reduce blood viscosity, content of fibrinogen, hemocyte aggregation and platelet aggregation.
应用推荐