紫外线诱导皮肤癌的机制是什么？
研究目的：第一部分探讨紫外线诱导真皮成纤维细胞凋亡的发生机理。
Objectives: Part I was to explore the mechanism of ultraviolet-induced fibroblast apoptosis.
目的：探讨表皮生长因子受体(EGFR)在紫外线诱导血管内皮生长因子(VEGF)分泌中的作用。
Objectives: to explore the role of epidermal growth factor receptor (EGFR) on UV-induced VEGF secretion of MEF.
目的探讨在紫外线诱导的人晶状体上皮细胞DNA损伤修复过程中端粒酶活性和氧化损伤蛋白的变化和作用。
Objective To investigate the changes and the role of telomerase activity and other stress-related proteins in the process of UV-induced DNA damage and repair in human lens epithelial cells.
化合物5对紫外线诱导的A375 -S2细胞死亡具有明显的抑制作用，且对该细胞没有显示出细胞毒活性。
Compound 5 shows the inhibitory effect on UV-induced A375-S2 cell death and no cytotoxic effect on the normal A375-S2 cells.
目的：观察紫外线照射诱导新生大白鼠表皮细胞凋亡。
Objective:To observe the induction of apoptosis by ultraviolet radiation in neonatal mice keratinocytes.
结论紫外线可诱导软骨细胞凋亡。
Conclusion Ultraviolet radiation could induce apoptosis of chondrocytes.
水稻叶片中的ALDH7B受稻瘟病菌感染、紫外线和机械损伤诱导表达，而未经处理的水稻器官中检测不到表达。
ALDH7B7 expression was induced by blast fungus infection, ultraviolet, mechanical wound in rice leaves and was not detected in untreated rice organs.
水稻叶片中的ALDH7B受稻瘟病菌感染、紫外线和机械损伤诱导表达，而未经处理的水稻器官中检测不到表达。
ALDH7B7 expression was induced by blast fungus infection, ultraviolet, mechanical wound in rice leaves and was not detected in untreated rice organs.
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