认为P＿3（或N＿3）是三叉神经根及三叉神经脊束核的突触前和突触后的混合电位。
It was not the rnuscle potential. We believe that P3(N3 )is mixedpresynaptic and postsynaptic potentials evoked from the trigeminal root and nucleus of trigeminalspine bundle.
在蛋白质到达后，这些突触失去了对到来的信息反应的能力，导致记忆丧失。
After the proteins have attached, the synapses lose their capacity to respond to incoming information, resulting in memory loss.
另外，在传出信息的神经末端会释放少量的神经传递素，从而增加突触后神经细胞中的神经胶质素。
In addition, the nerve endings from where information is sent must release slight amounts of neurotransmitters to stimulate the neuroligin in the postsynaptic nerve cell.
化学突触也经常用来放大神经信号，即便是一个很小的突触后细胞也能改变一个很大的突触后细胞对信号的响应。
Chemical synapses also serve to amplify neuronal signals, so that even a small presynaptic nerve terminal can alter the response of a large postsynaptic cell.
电信号到达传递神经元的突触附近后，释放化学物质包。
At the side of a synapse that belongs to the transmitting neuron, an electrical signal arrives and releases packets of chemicals.
每个神经突触都有拥有一系列蛋白，它们像机器的零件一样，结合在一起，搭建出一个被称为突触后致密区(postsynaptic density PSD)的分子机器。
Within each synapse is a set of proteins, which, like the components of an engine, bind together to build a molecular machine called the postsynaptic density - also known as the PSD.
这种用前肢踩踏的训练，一个小时内就可以在两侧运动皮层的椎体神经元上形成突触后“树枝状小棘”的结构。
Training in a forelimb reaching task leads to rapid (within an hour) formation of postsynaptic dendritic spines on the output pyramidal neurons in the contralateral motor cortex.
研究再一次显示，醒后较久与睡眠状态相比，脑对同水平刺激的反应明显增强，而这表明突触活动有增强。
The studies again showed that, for the same levels of stimulation, responses were stronger following a long period of waking and weaker after sleep, suggesting that synapses must have grown stronger.
它们的平均振幅是依赖精确的后突触ap与EPSP时间，相差性的上下控制。
Their average amplitudes were differentially up - or down-regulated, depending on the precise timing of postsynaptic APs relative to EPSPs.
模型方程分别对应于突触后电位、感受器电位、始段分级电位和轴突上的的脉冲系列，每个方程都具有明确的生理意义。
The equations of the model, corresponding to postsynaptic potentials, receptor potentials, initial segment graded potentials and the impulse train along the axon respectively, have been given.
第一，川楝素以先易化后抑制的双相作用干扰神经递质释放，阻遏神经肌肉接头和中枢神经突触的突触传递。
The first is that by interfering with neurotransmitter release by causing an initial facilitation, TSN eventually blocks synaptic transmission at both the neuromuscular junction and central synapses.
研究突触最大的困难在于如何区辨所观察到的反应是来自突触前或突触后因子所导致。
A major challenge in studying the synapse is how to tell what observed effects are due to presynaptic factors and which to postsynaptic ones.
目的探讨突触后密度蛋白- 93 (PSD - 93)在脊髓损伤(SCI)后的表达变化以及定位情况。
Objective To study the expression and distribution of postsynaptic density protein-93 (PSD-93) after spinal cord injury (SCI).
目的探讨短暂脑缺血再灌流后突触传递功能的改变和神经系统的可塑性。
Objective To investigate the synaptic transmission function and neural plasticity after transient ischemia followed by repufusion.
结果提示，背索突触后神经元具有相对简化的几何构筑和相当复杂的局部突触回路。
The results indicate that dorsal column postsynaptic neurons possess a relatively simplified geometric organization and a considerable complexity in their local synaptic circuitry.
因谷氨酸代谢异常，过度释放和对突触后受体的高频刺激，引起了兴奋性神经毒性，导致神经元死亡。
The abnormal metabolism and excess release of Glutamic acid as well as high-frequency stimulation to the postsynaptic receptor induce excitatory neural toxicity, resulting in the death of neurons.
应用透射电镜射像及图像分析技术观察电刺激对脑梗后突触形态及其结构参数的变化。
The synaptic interface structure and the changes of its indexes of rats after cerebral infarction were analyzed with transmission electron microscopy and computer image processing system.
现在开发这些突触后蛋白组给了研究者一个战略切入点，其他人则可以坐在前排位置，见证神经科学展开人类大脑疾病的内在奥秘。
Mining the postsynaptic proteome now gives researchers a strategic entry point, and the rest of us a front row seat to witness neuroscience unravel the complexity of human brain disorders.
目的探讨戊四氮诱导癫痫对大鼠空间学习记忆功能的影响及海马突触后致密物95（PSD-95）的表达变化。
Objective To explore the effect of pentylenetetrazol(PTZ)-kindled epilepsy on rats learning, memory and the expression of postsynaptic density 95(PSD-95).
突触后电位表现电流效应和频率依赖性，其IPSP具有长时程的抑制作用。
Postsynaptic potentials manifested current effect and frequency dependency and their IPSP displayed a prolonged inhibition.
突触前递质释放装置的发育成熟和突触后受体的转运是中枢神经系统突触发育过程中非常重要的两个方面。
Maturation of presynaptic transmitter secretion machinery and trafficking of postsynaptic receptors are two critical steps in synaptogenesis and synaptic development of central nervous system (CNS).
在树突发育和突触功能活性依赖的调节过程中，突触后致密蛋白磷酸化的机制仍然是未知的领域。
Phosphorylation of postsynaptic density proteins remains a largely unexplored mechanism in activity-dependent regulation of dendrite development and synaptic function.
以往研究认为其具有拟gaba样作用，通过增强突触后gaba_a受体介导的中枢抑制而产生麻醉作用。
The previous studies indicated it produced anesthetic effect through its mimic GABA action, which enhanced central inhibition mediated by postsynaptic GABAa receptors.
这些结果表明，该突变不仅影响集聚蛋白诱导突触后结构的功能，也影响神经肌肉接头功能的维持。
These results indicate that the mutation does not interfere with the ability of agrin to induce postsynaptic structures but that it dramatically perturbs the maintenance of the neuromuscular junction.
树突棘是神经元树突上的功能性突起结构，通常作为突触后成份与投射来的轴突共同构成完整的突触连接。
Dendritic spines are functional protrusions on neuron dendrites and generally act as postsynaptic sites to form integral synapses with axon terminals.
目的研究大鼠发育过程中视皮层神经元的突触后电流变化，探讨视皮层视觉依赖性突触的形成和重新分布的细胞内机制。
Objective To investigate the changes of post synaptic currents (PSCs) of neurons in developing visual cortex for intracellular mechanisms of experience dependent synapse formation and redistribution.
神经元的功能主要由突触后膜、胞体膜和始段膜三种膜结构的不同活动特性决定的。
The membrane of a neuron is mainly composed of the following three parts:the postsynaptic membrane, the somatic membrane and the membrane of the initial segment.
采用全细胞电压钳记录技术，给予选择性受体阻断剂以分离不同的突触后受体电流。
The recordings of postsynaptic currents were performed by using the whole-cell voltage-clamp method.
采用全细胞电压钳记录技术，给予选择性受体阻断剂以分离不同的突触后受体电流。
The recordings of postsynaptic currents were performed by using the whole-cell voltage-clamp method.
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