海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
目的观察大鼠脑梗死及电针干预后突触素(syp)的动态变化，探讨突触可塑性的物质基础、机制及针刺的影响。
Objective To explore the mechanism, material fundamental of synapse plasticity and acupuncture's efficacy by watching the alteration of SYP after cerebral infarction and acupuncture treatment.
已有广泛的研究表明突触可塑性如长时程增强和长时程抑制可以作为学习和记忆机制的细胞模型。
Extensive studies indicated that synaptic plasticity, such as long-term potentiation or long-term depression, is the cellular basis for learning and memory.
对影响认知的中枢胆碱能神经元的神经营养支持作用、抑制细胞凋亡、调节突触可塑性等可能是它们发挥保护作用的主要机制。
Neurotrophic factors can sustain the function of central cholinergic neurons as well as inhibit the neuronal apoptosis. They can also modulate the synaptic plasticity.
本课程探讨神经元传导，可激发细胞膜上之离子通道，神经突触传导与神经突触可塑性的分子以及细胞机制。
Surveys the molecular and cellular mechanisms of neuronal communication. Coversion channels in excitable membrane, synaptic transmission, and synaptic plasticity.
当开始了解这些突触改变的细胞生物学机制的时候，也应该考虑这种突触可塑性在完整大脑中的功能意义。
Yet, while we are beginning to understand the cellular mechanisms underlying synaptic changes, it is important to consider the functional implications of synaptic plasticity in the intact brain.
当开始了解这些突触改变的细胞生物学机制的时候，也应该考虑这种突触可塑性在完整大脑中的功能意义。
Yet, while we are beginning to understand the cellular mechanisms underlying synaptic changes, it is important to consider the functional implications of synaptic plasticity in the intact brain.
应用推荐