目的评估积雪苷霜治疗烧伤创面的疗效。
Objective To evaluate the efficacy of centella triterpenes cream in the treatment of burn wounds.
前言：目的：建立积雪苷霜软膏的质量控制方法。
Objective: To develop the quality standard of Jixuegan Shuang Cream.
目的：建立积雪苷片中积雪草苷和羟基积雪草苷含量的测定方法。
Objective:To establish a method for determining the contents of siaticoside and madecassoside in Jixuegan tablet.
[摘要]目的：探讨凹陷性痤疮瘢痕应用微等离子束联合积雪苷霜软膏治疗的临床价值。
Abstract: Objective To discuss the clinical value of atrophic acne scars with the way of micro-plasma radio-frequency combined with Centella Triterpenes Cream treatment.
积雪草苷对细胞周期和凋亡的影响。
方法：以积雪草苷提取率为评价指标，采用单因素实验与正交实验设计优选出积雪草中积雪草总苷的提取条件。
Methods: the extraction technique was evaluated by single factor experiment and orthogonal design, with the extraction rate of the asiaticoside as indicator.
目的采用薄层扫描法测定积雪甙片中积雪草苷的含量。
Objective to determine the content of asiaticoside in Jixuedai tablet by thin lay chromatography scanning (TLC-scanning).
目的探讨积雪草总苷元的抗抑郁作用及其可能作用机制。
OBJECTIVE To investigate the anti-depressed effects of Centella triterpenic genines and to explore its possible mechanism.
近年来经过研究发现，这些积雪草所含苷类的活性与苷元由直接的关系。
The activity of glycoside has direct relationship with aglycone in Centalla asiatica through the research in recent years.
羟基积雪草苷在大鼠肝微粒体中的代谢稳定性很高。
The metabolic stability of madecassoside in liver microsomes is good.
本研究结果对于进一步了解羟基积雪草苷在人体中的代谢和结构修饰具有重要的意义。
The result of this study has a great significance to further understanding of madecassoside in the metabolism and the structural modification of madecassoside.
目的研究积雪草苷对小鼠脓毒症致急性肝损伤的保护作用，并初步探讨其作用机制。
ObjectiveTo study the protective effect and mechanism of asiaticoside on sepsis-induced acute liver injury in mice.
我们通过复制经典的LPS致ALI动物模型，观察积雪草苷对LPS致ALI动物模型有无保护作用，其作用是否与影响炎症因子和细胞凋亡有关。
To observe whether the AS has the protective effects against ALI and possible mechanism related to inflammatory factors and apoptosis, we set up the model of ALI induced by LPS.
我们通过复制经典的LPS致ALI动物模型，观察积雪草苷对LPS致ALI动物模型有无保护作用，其作用是否与影响炎症因子和细胞凋亡有关。
To observe whether the AS has the protective effects against ALI and possible mechanism related to inflammatory factors and apoptosis, we set up the model of ALI induced by LPS.
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