他说：“胶质细胞增生发生在大脑中就相当于身体伤口的自动愈合，这种情况通常发生在出现神经元的损伤时，例如中风和多发性硬化症等等。”
He said: 'Gliosis is thought to be the brain equivalent of wound healing and is typically seen in conditions of neuronal injury, such as stroke and multiple sclerosis.
结果：毛果芸香碱惊厥持续状态模型鼠病理损伤特点为神经元坏死、细胞凋亡、凋亡蛋白表达增多，胶质细胞增生。
RESULTS: Neuron necrosis, apoptosis, increase of apoptosis protein expression and proliferation of glial cells were the characteristics of pathological injury in the pilocarpine induced se models.
大鼠模型脑出血后脑组织的改变包括血肿周围存在不同程度的水肿、炎性细胞浸润、神经元坏死及胶质细胞和血管增生。
The brain tissue changes in mouse model with brain hemorrhage, included dropsy in different degree, inflammatory cell infiltrate, nerve cell necrosis and vascular proliferation as well as gliacyte.
结论：视神经间接损伤后，早期适量的X射线局部照射能有效地抑制胶质细胞的反应性增生，能明显地促进神经轴突的结构再生。
It is suggested that early proper X-ray irradiation on the injured optic nerve can effectively inhibit the proliferation of astrocytes and obviously promote the axonal regeneration of optic nerve.
缺血后脑的病理改变以神经细胞变性、固缩和退变，皮层下白质的神经纤维和髓鞘溃变为主，普遍存在胶质细胞增生，形成胶质小结。
There were obvious pathological change in the brain after the chronic cerebral hypoperfusion, such as myelinic degeneration and formation of glial nodule in white matter.
模型组海马CA1区神经元数量明显少于空白组（P< 0 0 1 ） ，可见神经元脱失，并有胶质细胞增生。
The quantities of model group neuron were less than that of control group neuron ( P< 0.01) in the CA1 region of hippocampus, and the loss of neuron and hyperplasia of colloid cells could be observed.
结果在手术切除的病灶组织中，除一些增生性的星形胶质细胞同时具上述三种蛋白的阳性标记外，在一些异形的神经元内同时还存在有MDR1和MRP的阳性表达。
Results An over expression of immunoreactivities for MDR 1 and MRP was found at both reactive astrocyte and some giant dysplastic neurons, while the overexpression of GFAP co…
结果难治性颞叶癫痫患者颞叶组织胶质细胞呈不同程度增生，部分有噬神经元现象和神经元变性。
Results Gliosis was observed, to different extent, in the brain tissues from patients with refractory temporal lobe epilepsy, so were neuronophagia phenomenon and neuronal degeneration.
结果难治性颞叶癫痫患者颞叶组织胶质细胞呈不同程度增生，部分有噬神经元现象和神经元变性。
Results Gliosis was observed, to different extent, in the brain tissues from patients with refractory temporal lobe epilepsy, so were neuronophagia phenomenon and neuronal degeneration.
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