结论髓鞘内甘丙肽可能通过减低运动神经兴奋性而产生抑制屈肌运动效应，从而有利防止骨骼肌运动过度。
Conclusion the inhibitory effect of intrathecal galanin on flexor contractions is probably due to reduction in the excitability of motoneuron and might prevent muscles from over - movement.
因谷氨酸代谢异常，过度释放和对突触后受体的高频刺激，引起了兴奋性神经毒性，导致神经元死亡。
The abnormal metabolism and excess release of Glutamic acid as well as high-frequency stimulation to the postsynaptic receptor induce excitatory neural toxicity, resulting in the death of neurons.
结果：视网膜缺血时兴奋性氨基酸过度释放导致神经元死亡。
Results: The release of excess excitatory amino acids caused neuronal death in ischemic retina.
结果：视网膜缺血时兴奋性氨基酸过度释放导致神经元死亡。
Results: The release of excess excitatory amino acids caused neuronal death in ischemic retina.
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