海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
研究人员指出，酒精并不是通过杀死神经元细胞导致失忆发生的。实际上，是这些产生的甾体物质影响了突触可塑性，造成了LTP和记忆形成方面的损害。
The scientists point out that alcohol isn't causing blackouts by killing neurons. Instead, the steroids interfere with synaptic plasticity to impair LTP and memory formation.
腹侧被盖区多巴胺神经元的突触可塑性的改变可能在许多药物滥用(包括可卡因滥用)所导致的回路修饰中起着一定作用。
Changes in synaptic plasticity in dopamine neurons of the ventral tegmental area (VTA) may contribute to circuit modification induced by many drugs of abuse, including cocaine.
已有的电生理学实验已经表明，褪黑素可以调节海马神经元的兴奋性和突触可塑性。
Previous electrophysiological experiments have revealed that melatonin modulates hippocampal neuronal excitability and plasticity.
对影响认知的中枢胆碱能神经元的神经营养支持作用、抑制细胞凋亡、调节突触可塑性等可能是它们发挥保护作用的主要机制。
Neurotrophic factors can sustain the function of central cholinergic neurons as well as inhibit the neuronal apoptosis. They can also modulate the synaptic plasticity.
本课程探讨神经元传导，可激发细胞膜上之离子通道，神经突触传导与神经突触可塑性的分子以及细胞机制。
Surveys the molecular and cellular mechanisms of neuronal communication. Coversion channels in excitable membrane, synaptic transmission, and synaptic plasticity.
本课程探讨神经元传导，可激发细胞膜上之离子通道，神经突触传导与神经突触可塑性的分子以及细胞机制。
Surveys the molecular and cellular mechanisms of neuronal communication. Coversion channels in excitable membrane, synaptic transmission, and synaptic plasticity.
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