大脑中认知区域的神经元死亡增加。
Neuronal death increased in brain areas critical for cognition.
这是由神经元死亡导致的。
脑梗死时的神经细胞损伤以神经元死亡最为突出。
The neuronal death is most prominent in nerve cell damage during cerebral infarction.
兴奋毒性可能是造成神经元死亡的“最后公路”。
The excitotoxicity may be the last public way by which neurons were led to death.
结果：视网膜缺血时兴奋性氨基酸过度释放导致神经元死亡。
Results: The release of excess excitatory amino acids caused neuronal death in ischemic retina.
脑缺血时，神经元释放的谷氨酸是引起神经元死亡的重要原因。
Glumate (Glu) released from neurons during brain ischemia is a main cause of inducing neuron death.
实验证据表明，这种迟发的神经元死亡的分子机制中存在凋亡过程。
Several lines of evidence have been proposed to suggest that apoptotic process underlies the molecular mechanism of this delayed neuronal cell death.
在急性缺血过程中，会激发一系列导致神经元死亡的病理性瀑布反应。
A series of pathological cascades leading to neuronal death are triggered in acute ischemia.
我们现在认为可能并不是淀粉样纤维细丝导致神经元死亡和疾病的发作。
We now think that it may not be the amyloid fiber filaments that cause neuron death and disease onset.
给予外源性NGF可以减少迟发性神经元死亡，起到保护神经元的作用。
Delayed neuronal death can be reduced by providing NGF, which can protect neurons.
目的研究灯盏花素对沙土鼠缺血再灌注后海马延迟性神经元死亡的影响。
Objective To investigate the protective effect of erigeron on the delayed death of the hippocampal neurons after ischemia reperfusion.
目的：用重复脑缺血再灌注小鼠模型的海马切片观察迟发性神经元死亡的形态学特点。
Objective:To observe the morphological changes of delayed neuronal death in cerebral hippocampal sections of mice with repeated ischemia-reperfusion.
成年哺乳动物中枢神经系统(CNS)损伤，常导致神经元死亡及轴突不可逆的退化。
Damage to the central nervous system (CNS) is always followed by an irreversible axon degeneration of injured neurons.
因谷氨酸代谢异常，过度释放和对突触后受体的高频刺激，引起了兴奋性神经毒性，导致神经元死亡。
The abnormal metabolism and excess release of Glutamic acid as well as high-frequency stimulation to the postsynaptic receptor induce excitatory neural toxicity, resulting in the death of neurons.
目的：观察许旺细胞源神经营养因子对周围神经高位损伤所致脊髓背根节感觉神经元死亡的保护作用。
OBJECTIVE: To observe the protective effect of Schwann cell-derived neurotrophic factor on the high injury of peripheral nerve-induced apoptosis of sensory neurons in spinal dorsal root ganglia.
目的探讨淋巴滞留性脑水肿大鼠空间认知能力的改变及其与海马迟发性神经元死亡（DND）之间的关系。
Objective To investigate changes of spatial cognitive function and the relationship with delayed neuron death(DND)in hippocampus of lymphostatic cerebroedema in rats.
目的研究凋亡是否参与嗅上皮正常生理更替和嗅球摘除后嗅觉神经元死亡后再生过程，探讨凋亡与神经元再生的关系。
Purpose To study whether apoptosis play a role in controlling the number of olfactory receptor neurons, so as to reveal the speciality and its relationship in neurogenesis.
结论复灌开始后连续应用GM 1治疗对脑缺血造成的记忆障碍有明显改善作用，这可能与其减少迟发性神经元死亡有关。
Conclusion During recirculation treatment with GM1 exhibited a promising effect of improvement of memory impairment by BIR, and it may be related the reduction of DND.
由于铁可导致DA神经元死亡，因此铁含量的增加可能是DA含量减少的原因之一，甲磺酸去铁胺具有保护DA神经元的作用。
Elevation of iron content in SN is one of the mechanisms responsible for the reduction of DA content. Desferrioxamine mesylate may exert a protective action on dopaminergic neurons.
目的：围产期缺氧缺血性脑损伤是导致儿童神经系统发育障碍及后遗症的严重疾病，已知脑损伤后迟发性神经元死亡以凋亡为主。
Objective: Perinatal hypoxic-ischemic brain injury is an important cause of neurodevelopmental impairment and disability. It's well-known that the main death 'form of neuron after HIBD is apoptosis.
然而，关于这些斑块到底是老年性痴呆症中神经元死亡的起因，还是仅仅为这种疾病的一个副产物，科学家之间仍然存在相当大的争论。
There is still considerable debate among scientists as to whether these plaques are the cause of the neuronal death that occurs in Alzheimer's or just a by-product of the disease, however.
近年来认识到脑缺血在缺血性损伤过程中除缺氧和能量代谢衰竭外，由缺血诱导的一系列瀑布样效应是导致缺血性神经元死亡的重要机制。
Recently, it has been found that a series cascade damages induced by ischemia is the important mechanisms of ischemic neuron death, in addition to anoxia and energy failure.
帕金森氏病的典型特征是中脑某些区域的多巴胺神经元的死亡。
Parkinson's disease is characterized by the death of dopaminergic neurons in areas of the midbrain.
当我们中风时，大脑缺乏氧气，造成神经元细胞大批死亡，导致我们瘫痪在床并且无法说话。
When we have a stroke, our brain is starved of oxygen, causing the catastrophic death of those nerve cells and leaving us paralysed and unable to speak.
2011年1月4日在相邻两个神经元(亦称为神经突触)之间释放的锌离子是重要的传导信号，但是锌累积过多，会导致细胞失去功能甚至死亡。
January 4, 2011 zinc ions released at the junction between two neurons (called a synapse) are important signals, but when too much zinc accumulates, cells become dysfunctional or die.
在增殖期过后第二个三个月里，神经元也被“削减”，这意味着它们经历了一个有计划的细胞死亡过程。
After a period of proliferation during the second trimester, neurons are also "pruned," meaning that they undergo a planned cell death.
当脑细胞开始死亡时，首当其冲凋亡的就是那些参与学习以及记忆的神经元。
Among the first ones to go when brain cells start dying are those involved in learning and memory.
他们通过这个称为LRRK2基因的突变形式精确的了解到该蛋白的非正常功能，以及它是如何影响神经元并最终导致细胞死亡。
They have pinpointed the malfunction of the protein made by mutant forms of the gene called LRRK2 and how it affects neurons, ultimately leading to their death.
他们通过这个称为LRRK2基因的突变形式精确的了解到该蛋白的非正常功能，以及它是如何影响神经元并最终导致细胞死亡。
They have pinpointed the malfunction of the protein made by mutant forms of the gene called LRRK2 and how it affects neurons, ultimately leading to their death.
应用推荐