主要实验室检查包括血清钙、磷、碱性磷酸酶活性、甲状旁腺激素、尿磷和肝肾功能。
Main laboratory tests included serum calcium, phosphorus, alkaline phosphatase activity, parathyroid hormone, urinary phosphorus as well as liver and renal functions.
蛋白质激酶的磷酸化级联放大系统在激素的信号转导中起重要作用。
Signalling cascades consisting of protein kinases play important roles in hormone signal transduction pathways of plants.
目的：探讨糖皮质激素对肾病综合征患儿血清钙、骨源性碱性磷酸酶的影响，了解患儿成骨细胞功能变化。
Objective: to study the effect of prednisone to serum calcium and osteo-alkaline phosphatase of children with nephrotic syndrome and their functional changes of osteoblast.
已有证据显示PKA通过磷酸化脂周素调节脂解（13-17），引起构象改变使脂滴暴露给内源性的，激素敏感的脂肪酶（14）。
Evidence suggests that PKA regulates lipolysis by phosphorylating perilipin (13-17), resulting in a conformational change that exposes lipid droplets to endogenous, hormone-sensitive lipases (14).
结论：皮质激素治疗可显著抑制肾病综合征患儿的成骨细胞功能，导致血清钙及碱性磷酸酶水平的降低。
Conclusion: the corticoid can remarkably suppress the osteoblast function of children with nephrotic syndrome, leads to reduction of level of serum calcium and osteo-alkaline phosphatase.
磷酸葡萄糖脱氢酶（G6PD）是一个雌激素诱导酶，人乳腺癌较高雌激素受体水平与较高G6PD活性有关。
Glucose-6-phosphate dehydrogenase (G6PD) is an estrogen-mediated enzyme, In human breast carcinoma, the level of G6PD activity usually is correlated with the concentartion of estradiol receptors (ER).
分别于治疗前后检测每组病例血甲状旁腺激素（PTH）、骨特异性碱性磷酸酶（BAP）；
The levels of serum parathyroid hormone(PTH) and bone-specific alkaline phosphatase(BAP) were detected respectively before and after the treatment.
雌激素和阿仑磷酸钠治疗在组织学改变相似的情况下，阿仑磷酸钠由于抑制了破骨细胞的活性，其力学性能较雌激素替代治疗组有统计学意义上的提高。
With the similar histological change, alendronate treatment group, due to inhibition of the osteoclast cells, needs statistically greater pull out strength than estrogen treatment group does.
共12例基线时有粒细胞缺乏（<500/微升），10例接受了类固醇激素治疗，16例接受了钙调磷酸酶抑制剂或西罗莫斯治疗。
In all, 12 patients were neutropenic (<500/microl) at baseline, 10 received steroids and 16 calcineurin inhibitors or sirolimus.
雄激素受体基因突变、多种途径引起的异常磷酸化激活和共激活物的调节失控等是激素非依赖性前列腺癌形成的主要促成因素。
The mutation of androgen receptor gene, abnormal phosphorylation and abnormal regulation of co-activators contribute to the formation of androgen-independent prostate cancer.
该多克隆抗体通过用合成磷酸肽免疫动物制备，该合成磷酸肽是人糖皮质激素受体丝氨酸（211位）附近的残基。
Polyclonal antibodies are produced by immunizing animals with a synthetic phosphopeptide corresponding to residues surrounding serine 211 of human glucocorticoid receptor.
该多克隆抗体通过用合成磷酸肽免疫动物制备，该合成磷酸肽是人糖皮质激素受体丝氨酸（211位）附近的残基。
Polyclonal antibodies are produced by immunizing animals with a synthetic phosphopeptide corresponding to residues surrounding serine 211 of human glucocorticoid receptor.
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