GLRX2和TXN1 是人体细胞内有关氧化还原反应的基因,分别编码抗氧化酶系统成员硫氧还蛋白和非酶系统成员谷氧还蛋白。
GLRX2 and TXN1 are oxidation-reduction reactions related to human genes, coding antioxidase system member thioredoxin and non-enzymatic system member glutaredoxin respectively.
目的:从人脑组织中克隆硫氧还蛋白还原酶基因。
Objective: To clone the thioredoxin reductase (TR) gene from human fatal brain.
胞质硫氧还蛋白是一类保守的小蛋白,参与细胞氧化还原状态的调控。
Cytosolic thioredoxins are small conserved proteins that are involved in cellular REDOX regulation.
目的:利用硫氧还蛋白融合表达系统表达胃癌相关基因gcrg2 13。
Objective: To express gastric cancer related gene GCRG213 using thioredoxin fusion expression system.
结论线粒体型硫氧还蛋白还原酶基因和蛋白表达的增加有利于大鼠心肌抗氧化损伤。
Conclusions the increased expression of mitochondria thioredoxin reductase may protect the heart against oxidative stress injury.
结果应激组大鼠心肌中线粒体型硫氧还蛋白还原酶基因和蛋白表达均高于非应激组。
Results The expressions of the protein and gene of mitochondrial thioredoxin reductase in stress group were higher than those of control group.
下面就硫氧还蛋白系统的结构特点、功能,硫氧还蛋白系统与肿瘤的关系等研究进展作一综述。
This review provides a summary of advances about the structure, function of thioredoxin and the relationship between thioredoxin and tumors.
结论:用硫氧还蛋白融合表达系统在大肠杆菌中表达的小鼠内皮抑素重组融合蛋白易纯化并具有高活性。
CONCLUSION: The mouse endostatin recombinant fusion protein expressed in E. coli by using thioredoxin fusion expression system is easy to be purified and possesses high activity.
GLRX2和TXN1是人体细胞内有关氧化还原反应的基因,分别编码抗氧化酶系统成员硫氧还蛋白和非酶系统成员谷氧还蛋白。
GLRX2 and TXN1 are oxidation-reduction reaction related human genes, coding antioxidase system member thioredoxin and non-enzymatic system member glutaredoxin respectively.
GLRX2和TXN1是人体细胞内有关氧化还原反应的基因,分别编码抗氧化酶系统成员硫氧还蛋白和非酶系统成员谷氧还蛋白。
GLRX2 and TXN1 are oxidation-reduction reaction related human genes, coding antioxidase system member thioredoxin and non-enzymatic system member glutaredoxin respectively.
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