目的研究血管活性肽在血管再狭窄形成中的作用。
Objective To investigate the effects of several vasoactive peptides on the development of restenosis.
血管介入术后的再狭窄严重影响术后远期疗效。
Restenosis after vascular intervention influences the long term curative effect seriously.
血管成形术后发生再狭窄影响手术的远期疗效。
Restenosis after angioplasty influences the long dated surgical effect.
目的：观察辐射对血管活性物质的影响及其机制、意义以探讨辐射防治再狭窄的机制。
Objective: To study the effects and mechanism of radiation on various kinds of vasoactive substances.
血管内放射导致细胞死亡的主要形式为凋亡，电离辐射预防再狭窄的主要机制是平滑肌细脑的增殖抑制。
Apoptosis is a major style of cell death induced by endovascular radiation. The inhibition of SMCs proliferation induced by radiation is a main mechanism for prevention of restenosis.
目的：研究化痰益气活血治法对血管狭窄模型兔再狭窄相关基因表达的影响。
Objective:Influence of dissipate phlegm, invigorate vital energy, promoting blood flow treatment to restenosis related gene of rabbit angiostegnosis model was studied.
目的计算放射性球囊治疗冠状动脉再狭窄时导管内核素对正常血管的剂量分布。
Objective To estimate dose distribution in normal vessels delivered by radioactive solution in the catheter for intracoronary intra-balloon brachytherapy.
目的：探讨在不锈钢冠状动脉支架上携带质粒基因，为心血管再狭窄基因治疗的临床应用提供试验依据。
Objective: to investigate stainless steel coronary stent-based plasmid gene delivery system and evaluate its feasibility and effectiveness to gene therapy of cardiovascular restenosis.
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
目的评价兔髂动脉内近距离治疗预防经皮经腔血管成形术（PTA）后再狭窄的可行性及作用。
Objective To evaluate the role of intra arterial brachytherapy to prevent the iliac artery restenosis in the rabbit following percutaneous transluminal angioplasty (PTA).
本文综述了声动力疗法在肿瘤治疗、血管成形术后再狭窄等增殖性疾病方面的医学应用及相应的生物学机制。
This paper reviews the medical application of sonodynamic therapy in the treatment of tumor and restenosis after percutaneous transcoronary angioplasty and the corresponding biomechanism.
目的：探讨在血管再狭窄形成过程中内膜增生及血管重塑的作用。
AIM: To explore the role of intimal hyperplasia and vascular remodeling in the formation of vascular restenosis.
目的：评估质粒介导下蛋白涂层金属支架局部转染血管内皮生长因子(VEGF)基因对小型猪冠状动脉再狭窄的作用。
Objective: to assess the effects of local vascular endothelial growth factor (VEGF) transfer using protein coated metallic stents on restenosis of coronary arteries in mini swine model.
P 27蛋白的高表达可显著抑制血管平滑肌细胞增殖，有望成为治疗PT CA后再狭窄的新手段。
P27Kipl restrains the proliferation of VSMC obviously and is prospective to become a new means to cure restenosis after PTCA.
目的观察中药对冠心病支架术后缺血心肌血管新生以及支架再狭窄的临床疗效。
ObjectiveTo observe the effect of Tongxinfang on coronary collateral formation and stent-restenosis in patients with coronary artery disease after stenting.
防治静脉移植物再狭窄的方案包括药物治疗、基因治疗和血管外支架。
Therapeutic strategies to prevent vein graft disease include pharmacotherapy, gene therapy and external stenting.
同时，烟碱还有促进动脉粥样硬化和促进血管内膜增殖的作用，因此有促进再狭窄的作用。
Nicotine can also accelerate atherosclerosis and intimal proliferation, so may contribute to the development of restenosis.
血管成形术是治疗血管闭塞性疾病的主要手段，但术后再狭窄的高发生率为其的应用带来了阻碍。
Angioplasty is the main method of treating vascular occlusive diseases, but high incidence of restenosis after operation has hampered its clinical applications.
术后靶血管管腔再狭窄是冠状动脉介入治疗术的主要并发症，如何有效预防再狭窄已成为目前介入心脏医学界的主要课题之一。
The in-stent restenosis is one of the main complications in coronary artherosclerotic heart disease patients after PTCA and PCI.
而置入支架的血管可因一种叫做再狭窄的过程而再次闭塞，这就意味着要二次手术。
The artery in which the stent is placed, however, can become blocked again during a process called restenosis. This re-closure means a repeat surgery.
结论：局部转染vegf165基因可抑制血管新生内膜增生及血管再狭窄，为将来血管内膜增生的基因治疗奠定基础。
CONCLUSION: Local transfection of VEGF165 gene restrains intimal hyperplasia and restenosis of vessels, which lays a foundation for future gene therapy of vascular intimal hyperplasia.
结论 女性、既往CABG病史、CTO病变、血管直径、置入支架长度是支架术后再狭窄的危险因素，而糖尿病史等与再狭窄无关。
ConclusionFemale, CABG history, CTO lesion, vessel size and length of stent are associated with followup restenosis, while traditional risk factors such as diabetes mellitus have no impact on ISR.
冠心病危险因素、血管造影下冠状动脉病变特征、支架术操作技术参数是影响冠脉病变支架术后再狭窄的主要危险因子。
The risk factors of coronary artery disease, angiographic and procedural features are the main risk factors of restenosis after coronary artery stent implantation.
目的血管腔内支架成形术目前已被证明是治疗外周动脉硬化闭塞症（PAOD）的有效方法，然而术后出现的再狭窄影响了支架成形术的疗效。
Objective:It is proved that Percutaneous Transluminal Angioplasty and Stenting is a effective treatment of PAOD, but its benefits is seriosly limited by local thrombotic occlusion and restenosis.
当该支架用于治疗血管损伤时，可以很好的防止临床再狭窄，即使血管损伤程度涉及血管过度拉伸在直径上超过30%也可以做到。
The stent, when used to treat a vascular injury, gives good protection against clinical restenosis, even when the extent of vascular injury involves vessel overstretching by more than 30% diameter.
药物治疗是减少冠心病介入术后支架内再狭窄、降低远期主要心血管事件的主要手段。
Medicine treatment plays key role in reducing the rate of restenosis and major adverse cardiac events.
结论 血管内超声是评价支架内再狭窄的有效的手段， 支架内再狭窄患者有更大的斑块面积和内膜增生面积，而最小管腔面积变小。
Conclusion IVUS is the effective ways to access the ISR, the larger plaque area, neointimal area but smaller minimal lumen area were seen in patients with ISR.
因此，降低血管受损局部基质金属蛋白酶表达将成为防治血管内再狭窄的一个方向。
Therefore, reduction of MMPs expression in the local area after vascular injury provides a direction for preventing and curing vascular restenosis.
术后6个月重复冠状动脉造影，比较两组间的晚期管腔丢失、支架再狭窄率及术后心血管不良事件发生率。
Coronary angiography was performed at the 6th month. The late lumen loss, stent restenosis and major adverse cardiac events were compared in both groups.
当血管损伤后，局部产生的细胞因子引起炎症反应是血管再狭窄的主要原因。
The vascular restenosis is attributable to a local inflammation after vascular injury, which results in the production of cytokines.
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