术后晶状体囊残留的晶状体上皮细胞的增殖、迁移、纤维化生是形成后发性白内障的主要原因。
PCO is caused by the lens epithelial cells retained in the capsular bag following surgery, which proliferate, migrate and transform to myofibroblasts.
理论上说，使用药物彻底清除残留于晶状体囊袋内的晶状体上皮细胞将是预防后发性白内障最直接的方法。
Theoretically, clearance of the remaining lens epithelial cells within the capsular bag with medicine is the most effective way to prevent PCO.
目的：研究不同浓度的高糖对人晶状体上皮细胞凋亡的作用。
AIM: to investigate the effect of high glucose with various concentration on apoptosis of human lens epithelial cells.
目的探讨老年性白内障晶状体上皮细胞凋亡的生化特征。
ObjectiveTo investigate the biochemical characteristics of senile cataract lens epithelial cell apoptosis.
目的探讨热休克蛋白(HSP)27在老年性白内障晶状体上皮细胞中的表达及意义。
Objective To investigate the expression of heat shock protein 27 (HSP27) in human age related cataract lens epithelial cells.
目的：为探讨热休克蛋白- 70对人类糖尿病性白内障晶状体上皮细胞的影响。
AIM: To study the effect of heat shock protein-70 (HSP-70) on lens epithelial cells (LECs) in human diabetic cataract.
白内障是最常见的致盲眼病之一，晶状体上皮细胞的凋亡是除先天性白内障以外的所有类型白内障发生的共同细胞学基础。
Cataract is one of the most common eye diseases causing blind, lens epithelial cell apoptosis is the cellular bas is of all kinds of cataract except congenital cataract.
基质金属蛋白酶-2,9及其抑制因子- 1,2在老年性白内障晶状体上皮细胞的表达。
Expression of Matrix Metalloproteinase-2, -9 and Their Inhibitors TIMP-1, -2 on Lens Epithelial Cells of Aged-related Cataract.
中药制剂的实验药理研究主要集中在抗氧化作用、抑制醛糖还原酶作用和抑制晶状体上皮细胞凋亡方面。
Pharmacological experiments of Chinese herbal medicine were focused on antioxidant effect and inhibiting aldose reductase and decreasing apoptosis of lens epithelial cell.
近些年的研究表明：多种调控基因参与了晶状体上皮细胞凋亡。
Recent studies have shown that a variety of regulatory genes involved in lens epithelial cell apoptosis.
人晶状体上皮细胞系内肌醇1，4，5-三磷酸受体和兰尼碱受体的表达和作用。
Expression and role of inositol 1,4,5-trisphosphate receptor and ryanodine receptor in a human lens epithelial cell line.
目的建立钝挫伤白内障大鼠模型及探讨钝挫伤白内障与其晶状体上皮细胞凋亡的关系。
Objective to build contusion cataract model of rats and observe the relationship between contusion cataract and lens epithelial apoptosis.
目的克隆死亡因子相关基因15（MRG15）并研究其在正常晶状体和年龄相关性白内障晶状体上皮细胞中的表达，比较二者的差异。
ObjectiveTo clone the gene of mortality factor related gene 15(MRG15) and to investigate its expression in lens epithelium cell(LEC) of normal and age related cataractous eyes.
残留晶状体上皮细胞在后囊的增殖、移行、纤维化生是后囊混浊形成的重要因素。
The opacification is mainly due to the proliferation and migration of postoperative remnants of lens epithelial cells on the posterior lens capsule.
后囊混浊是由于术后位于晶状体前囊及赤道部的上皮细胞增殖，并迁移至无细胞的后囊所致。
In PCO, residual LECs in the anterior and equatorial regions of the lens capsule after surgery proliferate and migrate onto the previously acellular posterior capsule.
但是迄今硕士研究生学位论文为止，对钝挫伤白内障发病机制的研究尚不多见，也未见到其晶状体上皮细胞凋亡超微结构的明确报道。
But till up now it was rarely known for the mechanism of contusion cataract, so was the accurate report for the ultrastructure of lens epithelial cells.
目的研究去整合素对体外培养的人眼晶状体上皮细胞生长的抑制作用。
Objective to study the inhibitory effect of disintegrin on adhesion and migration in cultured human lens epithelial cells (LECs) in vitro.
目的探讨在紫外线诱导的人晶状体上皮细胞DNA损伤修复过程中端粒酶活性和氧化损伤蛋白的变化和作用。
Objective To investigate the changes and the role of telomerase activity and other stress-related proteins in the process of UV-induced DNA damage and repair in human lens epithelial cells.
结论H PM可导致晶状体损伤及白内障发生，辐射引起上皮细胞增殖异常是白内障发生的重要原因。
Conclusions HPM may cause lens injury and cataract formation. HPM-induced proliferation disorder in lens epithelial cells may possesses a critical role in cataract development.
目的观察晶状体前囊膜下上皮细胞增殖的组织病理学变化，并探讨其发生原因。
Objective to observe the histopathological changes of lens subcapsular epithelial proliferation and analyze their etiology.
结果CLC - 3阳性表达于晶状体上皮细胞的细胞膜。
Results the positive expression of CLC-3 was distributed in membrane of the lens epithelial cells.
目的从细胞水平探讨后囊混浊形成的机理，及各种眼组织对晶状体上皮细胞的作用。
Objective To study the pathogensis of lens posterior capsular opacification and the effects of different ocular tissue on bovine lens epithelial cells(BLEC) in cell culture level.
结论高场强大剂量的EMP辐射可抑制晶状体上皮细胞的正常增殖。
Conclusion High power EMP radiation inhibited proliferation of lens epithelial cells.
前言：目的：观察表皮生长因子(EGF)在人胎儿晶状体上皮细胞的表达。
Objective: to observe the expression of epidermal growth factor (EGF) in lens epithelial cells (LECs) of human fetus.
目的：欲研究白内障病人晶状体上皮细胞是否存在DNA损伤及损伤的程度。
Objective: To investigate the DNA damage in the lens epithelial cells in cataract patients.
结论：EGCG可能通过调控PK B的磷酸化水平而抑制晶状体上皮细胞的增殖，并不影响总蛋白含量。
CONCLUSION: EGCG could inhibit lens epithelial cell proliferation by regulating PKB phosphorylation - level, and EGCG has little effect on the total kinase protein.
结果0 .2 5 %、0 .2 %胰蛋白酶溶液处理的晶状体前囊，晶状体上皮细胞可以完全脱落。
Results 0.25%? 0.2% trypsin can clean up lens epithelial cells from anterior capsules totally.
多年来国内外学者一直致力于通过药物等方法抑制晶状体上皮细胞增殖的研究。
For many years, many scholars of domestic and overseas had been committed to study the methods to suppress the proliferation of lens epithelial cells by drugs.
目的观察体外培养人胚晶状体上皮细胞的生物学特性。
To investigate the biological characteristics of human embryonic lens epithelial cells cultured in vitro.
目的观察体外培养人胚晶状体上皮细胞的生物学特性。
To investigate the biological characteristics of human embryonic lens epithelial cells cultured in vitro.
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