白血病细胞的生存较正常细胞更好，随着时间的推移，它们会逐步取代正常细胞。
The leukemia cells grow and survive better than normal cells and, over time, they crowd out normal cells.
对儿童白血病和淋巴瘤的治疗提高了患者的生存几率。然而，有些治疗可能导致显著的长期或延迟效应。
Treatments for childhood leukemia and lymphoma have led to increased survival rates. However, some treatments may cause significant long-term or late effects.
结论12g5可改变HL 60细胞的生物学特性，从而在一定程度上抑制白血病细胞的增殖，影响细胞生存。
Conclusion 12g5 can change the biological properties of HL-60 cells and inhibit proliferation and survival of leukemia cells at a certain level.
目的探讨儿童急性淋巴细胞白血病(all)免疫表型与无病生存期(EFS)的关系。
Objective To investigate the relationship between immunophenotype and event-free survival (EFS) in children with acute lymphoblastic leukemia (ALL).
研究结果显示儿童急性淋巴性白血病的生存机会的减少与工频电磁场辐照有很强的关联。
Results of the study show a significant association between power-frequency magnetic field exposure and reduced disease-free survival for children with acute lymphoblastic leukemia.
目的对15 8例儿童急性淋巴细胞白血病（ALL）患儿治疗结果进行分析，探讨如何进一步提高儿童ALL无事件生存（EFS）率。
Objective To analyse the treatment outcome of 158 childhood acute lymphoblastic leukemia (ALL) patients, and explore how to improve the event-free survival(EFS) rate in ALL.
【译】结合在细胞遗传学正常的急性髓细胞性白血病的分子和复发和生存的预后指标。
Combined Molecular and Clinical Prognostic Index for Relapse and Survival in Cytogenetically Normal Acute Myeloid Leukemia.
目的：总结湘雅医院近年住院治疗儿童急性淋巴细胞性白血病(all)临床资料，探讨影响儿童all长期无病生存(DFS)的因素。
Objective: to analyse the treatment outcome of childhood acute lymphoblastic leukemia (ALL) patients in our hospital, and explore the factors influencing the Disease-free survival (DFS).
难治性急性白血病对治疗反应差，诱导缓解率低，复发率高，生存期短，是白血病治疗中的难题，目前仍以联合化疗为主要治疗方法。
Bad reaction to treatment, low induced relieving rate, high recurrence rate, and short survival period are tough problem in the treatment of refractory acute leukemia (RAL).
目的探讨生存素在化疗药物足叶乙甙(VP16)诱导白血病细胞凋亡中的作用。
Objective to discuss the role of survivin in the induction of apoptosis by VP16 in leukemia cells.
柳叶刀上有报道说如果供者的血和病人的匹配很好，那么两种方法的白血病的无病生存率很相近。
If the donor blood was well matched to the patient, the rate of leukemia-free surial was similar in both methods, according to the findings published in The Lancet.
慢性淋巴细胞性白血病的细胞依赖生存分子Bcl - 2，该分子可以关闭了癌症细胞的凋亡信号。
CLL cells are strongly dependent on a survival molecule called Bcl-2, which switches off self-destruct signals in the cancer cells.
30%的急性髓系白血病(aml)患者发生FLT3内部串联重复(itd)和酪氨酸激酶域(TKD)突变，而且生存预后较差。
FLT3 Internal tandem duplication (ITD) and tyrosine kinase domain (TKD) mutations are seen in 30% of AML patients (PTS) and are associated with poor survival.
30%的急性髓系白血病(aml)患者发生FLT3内部串联重复(itd)和酪氨酸激酶域(TKD)突变，而且生存预后较差。
FLT3 Internal tandem duplication (ITD) and tyrosine kinase domain (TKD) mutations are seen in 30% of AML patients (PTS) and are associated with poor survival.
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