根据近年的研究进展，从癌基因与抑癌基因的突变及环境因素的影响等方面综述了癌症的发生机理。
The occurrence mechanisms on cancer including mutation of oncogene and antioncogene, effect of environmental factors were reviewed.
其发生机制可能与肿瘤血管生成受阻、癌基因与抑癌基因、宿主免疫状态、肿瘤细胞增殖周期及信号传导有关。
The mechanism of tumor dormancy is related to hindrance of angiogenesis, oncogene and antioncogene, immunocompetence, proliferation cycle, signal conduction and so on.
越来越多的证据表明，肿瘤的发生是多因素作用的结果，与癌基因的激活与抑癌基因的失活有关。
More and more evidence indicated that the formation of tumor is due to multi factors with oncogene activation and anti oncogene inactivation.
热休克蛋白可能有助于细胞周期调控通过与原癌基因的产品或抑癌基因的产品。
Heat Shock Proteins may contribute to cell cycle regulation by interacting with proto-oncogene products or tumor suppressor gene products.
目前在分子水平上的研究显示肿瘤的发生与癌基因的激活和抑癌基因的失活以及细胞周期调节失控密切相关。
It has been proved that the occurrence of oncogenesis is in close relationship with the oncogene activation, the tumor suppressor gene inactivation and the disorder of cell cycle modulation.
肿瘤形成与控制细胞生长和分化的多种基因特别是癌基因、抑癌基因的遗传改变有关。
There are multiple genes especially oncogenes and tumor suppressor genes involved in 'the genetic alterations which regulating the growth and differentiation of cells.
研究表明几乎一半人类肿瘤都存在抑癌基因的失活，可见抑癌基因失活与肿瘤生长有着密切的关系。
Studies have sh own that the anti oncogene inactivation was closely related to tumor development because of the findings of anti oncogene inactivation in nearly one half of human tumors.
目的探讨胃癌组织中甲基化酶、去甲基化酶基因与肿瘤相关癌基因和抑癌基因的关系。
Objective To investigate the relationship of the expression of DNA methyltransferase, demethylase(mbd2) and tumor-associated genes in human gastric cancer.
癌基因的激活以及抑癌基因的失活被认为与细胞的恶性转化密切相关。
Transformation of cells is closely related to the inactivation of tumor-resistant genes and the activation of oncogenes.
这太有趣了，肿瘤基因（抑癌基因和原癌基因）的表达与多细胞生物体在遗传发生层面的形态协同一致的。
It is of interest that the coordinated expression of "cancer genes" (tumor supressors and oncogenes) coincides with the appearance of multicellularity in the filogenetic scale.
而肿瘤的发生与抑癌基因和癌基因密切相关。
And the occurrence of tumor is closely related to oncogenes and anti-oncogenes.
目的探讨癌基因MMP - 9和抑癌基因RECK在喉癌组织中的表达与喉癌临床特征的关系，了解喉癌的生物学行为，并研究两种基因的表达相关性。
ObjectiveTo investigate the relation between expression of MMP-9 and RECK in laryngeal carcinoma and it's clinical characteristics and to explore the biological action of laryngeal tumor.
目的：探讨癌基因P2 1和抑癌基因P16的表达与膀胱癌生物学行为之间的关系。
Purpose:Find the relationship between the expressions of P21 oncogene, P16 antioncogene and the biological action of the bladder cancer.
目的：探讨癌基因P2 1和抑癌基因P16的表达与膀胱癌生物学行为之间的关系。
Purpose:Find the relationship between the expressions of P21 oncogene, P16 antioncogene and the biological action of the bladder cancer.
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