结论：瘦素受体基因外显子4中519位点变异不是肥胖症的遗传因素。
CONCLUSION: Variation of exon 4 in LEPR gene at site 519 is not a genetic factor of obesity.
结论单纯型肥胖儿童瘦素受体基因第20外显子存在基因多态性的变化，且这种变化明显影响肥胖儿童的脂质代谢及体脂分布。
Conclusion There is polymorphism at 20 exon LEPR gene in children with obesity, which may affect the lipid metabolism and the fat distribution.
目的观察瘦素对大鼠肝细胞瘦素受体蛋白、基因表达及酪氨酸磷酸化的影响。
Objective to investigate effects of leptin on gene and protein expression and tyrosine phosphorylation of leptin receptor in rat hepatocytes.
目的探讨血瘦素水平和瘦素受体（LR）基因多态性对胰岛素敏感性影响及与缺血性脑血管病发病的关联。
Objective To study the effects of plasma leptin level and leptin receptor(LR) gene polymorphism on the insulin sensitivity and their relation to ischemic stroke.
瘦素是肥胖基因的表达产物，通过与其受体结合发挥其生物学作用。
The leptin is a expression product of the obesity gene. Biological effect of leptin-has appeared by combination with its receptor just.
目的观察瘦素对大鼠肝细胞瘦素受体蛋白、基因表达及酪氨酸磷酸化的影响。
The role of adhesion moleculesinduced tyrosine phosphorylation proteins in procollagen expression in fibroblasts;
目的观察瘦素对大鼠肝细胞瘦素受体蛋白、基因表达及酪氨酸磷酸化的影响。
The role of adhesion moleculesinduced tyrosine phosphorylation proteins in procollagen expression in fibroblasts;
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