川崎病的特征为广泛的中小血管炎症，以心血管系统的损害最为严重，可形成冠状动脉扩张和冠状动脉瘤。
Kawasaki disease is featured by wide polyangitis, especially that of cardiovascular system is most severe, which can form coronary arteriectasis and coronary aneurysm.
麻省理工学院生物与机械工程学塞西尔与艾达·格林（Cecil and Ida Green）杰出教授卡姆说，“市场上没有一种药能说明癌细胞如何挣脱原来的瘤、进入血管系统、迁出并形成继发瘤。
There isn't a single drug currently on the market that addresses how cancer cells break loose from a primary tumor and get into the vascular system, migrate out, and form a secondary tumor.
目的研究化疗对小儿神经母细胞瘤的VEGF表达以及血管形成状态的影响。
Objective to evaluate the effect of chemotherapy on VEGF expression and tumor microvessel formation in neuroblastoma in children.
血管炎症或许是一个促进动脉粥样硬化和动脉瘤形成的因素。
The vascular inflammation might be a factor that promotes atherosclerosis and the formation of aneurysms.
瘤怎样分泌增长系数在它的附近形成新血管，这为它提供它需要，为了增长的营养物，第2章描述。
Chapter 2 describes how tumor secretes growth factors to form new blood vessels in its vicinity, which provide it with nutrients it needs in order to grow.
新生血管大量形成在实体瘤的生长和转移中起着关键的作用。血管内皮生长因子(VEGF)是介导肿瘤血管生成的最主要因素。
New vessel formation plays a key role in tumor growth and transforming, and the Vascular endothelial growth factor (VEGF) is the most important factor inducing tumor vessel formation.
恶性瘤细胞侵袭血管是形成肿瘤转移的重要环节。
Invasion of malignant cells to endothelium is the major part of metastasis.
结论进展期小儿神经母细胞瘤患儿的术前化疗可抑制其血管形成，改善预后。
Conclusions the preoperative chemotherapy in children with advanced neuroblastoma can inhibit the tumor angiogenesis. It is related to a better prognosis.
川崎病的特征为广泛的中小血管炎症，以心血管系统的损害最为严重，可形成冠状动脉扩张和冠状动脉瘤。
Kawasaki disease(KD) is featured by wide polyangitis, especially that of cardiovascular system is most severe, which can form coronary arteriectasis and coronary aneurysm.
说明肥大细胞可能参与血管瘤的形成，并与血管内皮的增生有关。
It is suggested that mast cells are involved in the pathogenesis of hemangioma and the proliferation of endothelial cells.
对左胸廓内动脉行选择性动脉造影术，显示了一根分支血管的假动脉瘤形成。
Selective arteriography of the right internal thoracic artery revealed a feeder vessel for the pseudoaneurysm.
在分化的过程中上述组织的任何异常都可以形成肿瘤，发生血管瘤或(和)淋巴管瘤。
In the process of differentiation of the organization of any anomalies can form tumor, hemangioma or (and) lymphoceles.
结论弥漫性大B细胞性淋巴瘤治疗后生存期可通过肿瘤微环境中的免疫细胞、纤维化、血管形成的差异而影响。
Conclusions Survival after treatment of diffuse large-B-cell lymphoma is influenced by differences in immune cells, fibrosis, and angiogenesis in the tumor microenvironment.
肿瘤细胞表达基质金属蛋白酶类物质和勃附分子说明了恶性黑色素瘤组织存在血管生成拟态形成的物质基础。
TIMP, MMP-2,1C AM were secreted by the tumor cells can boost the adhere of tumor cells and matrix remold and turn into the substance base of vasculogenic mimicry .
肿瘤细胞表达基质金属蛋白酶类物质和勃附分子说明了恶性黑色素瘤组织存在血管生成拟态形成的物质基础。
TIMP, MMP-2,1C AM were secreted by the tumor cells can boost the adhere of tumor cells and matrix remold and turn into the substance base of vasculogenic mimicry .
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