结论：P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞端粒酶活性。
Conclusion: P53 protein significantly inhibits telomerase activity in human keloid fibroblasts.
结论P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞MMP1、MMP3蛋白的表达。
Conclusion P53 can obviously inhibit MMP1 and MMP3 expression in human keloid fibroblasts.
方法：取瘢痕疙瘩和正常皮肤，采用组织块培养法进行成纤维细胞体外培养。
METHODS: Keloid and norm al skin tissue were collected for the fibroblasts in vitro using tissue-block cultivation.
结论：几丁糖对正常皮肤及增生性瘢痕和瘢痕疙瘩来源的成纤维细胞的生长增殖均有抑制作用，有望在瘢痕的防治中发挥重要作用。
Conclusion Chitosan can inhibit the growth, proliferation of fibroblasts derived from keloid, hypertrophic scars and normal skin, and may play important role in the prevention and treatment of scars.
方法取新鲜瘢痕疙瘩组织，体外培养瘢痕成纤维细胞。
Methods: keloid fibroblasts were isolated from fresh keloid tissue and cultured.
免疫组化染色显示瘢痕疙瘩中成纤维细胞有丰富的I型前胶原表达。
Immunohistochemical staining showed increased expression of new formed, type I procollagen in keloid tissue.
免疫组化染色显示瘢痕疙瘩中成纤维细胞有丰富的I型前胶原表达。
Immunohistochemical staining showed increased expression of new formed, type I procollagen in keloid tissue.
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