目的：探讨治疗瘢痕疙瘩的方法。
目的了解瘢痕疙瘩遗传学改变的特征。
Objective To identify the genetic alteration in human keloid.
结论生存素可能参与瘢痕疙瘩的发病。
Conclusion Survivin might play a role in the development of keloid.
方法用透射电镜观察瘢痕疙瘩的超徽结构。
Methods The ultrastructure of keloid was observed by transmission electron microscope.
是一种较理想的建立瘢痕疙瘩模型的方法。
So this model is an ideal one for the studies of human keloids.
目的总结YAG激光治疗耳垂瘢痕疙瘩经验。
Objective To summarize the experience for treatment of earlobe scar with YAG-laser.
结论：曲安奈德注射液能够有效治疗瘢痕疙瘩。
Conclusion: Keloid can be effectively treated with triamcinolone acetonide.
目的探索瘢痕疙瘩误诊误治的原因，提高治疗效果。
Objective to explore causes for misdiagnosis and mistreatment of keloid in order to improve curative effect.
方法：我们临床上将瘢痕疙瘩分为惰性类和活跃类。
Methods We divided keloid into inertia type and active type clinically.
目的：探讨瘢痕疙瘩的综合疗法，并评价其临床效果。
Objective To explore the combined treatments for cheloid and evaluate their clinical effect.
方法取新鲜瘢痕疙瘩组织，体外培养瘢痕成纤维细胞。
Methods: keloid fibroblasts were isolated from fresh keloid tissue and cultured.
结论手术联合术后早期放疗是治疗瘢痕疙瘩的有效方法。
Conclusion Operation combined with early postoperative radiotherapy is a effective method to treat keloid.
目的：观察曲安奈德注射液瘢痕内注射治疗瘢痕疙瘩的疗效。
Objective: to observe the effect of injective triamcinolone acetonide for the treatment of keloid.
病理性瘢痕在临床上可分为增生性瘢痕和瘢痕疙瘩两种类型。
Pathological scars may be divided into two kinds: hypertrophic scars and keloids.
方法：对30例瘢痕疙瘩患者液氮冷冻后外涂美比欧疤痕平。
Method: 30 cases of keloid were treated with MEBO Scar Lotion after liquid nitrogen refrigeration.
在瘢痕疙瘩组织边缘，CTGF表达呈现由强至弱的过渡现象。
Transitional phenomenon was observed from strong to weak expression of CTGF protein at the edge of keloid tissue.
结论：皮损内注射瘢痕疙瘩方法简单，操作易掌握，疗效肯定。
Conclusion: The method of injected in lesions to cure keloid is simple, easy to master and is of positive efficacy.
目的：为了探讨美比欧疤痕平防治瘢痕疙瘩冷冻后的复发作用。
Objective: To investigate the efficacy of MEBO Scar Lotion in treating recurrent keloid after liquid nitrogen refrigeration.
结论：P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞端粒酶活性。
Conclusion: P53 protein significantly inhibits telomerase activity in human keloid fibroblasts.
结论：曲安奈得联合肤疾宁帖膏治疗瘢痕疙瘩效果好、疗效可靠。
Conclusion: Triameinolone acetonide combined Fujining is safe and effective for treating keloid.
目的：正确处理开放性伤口，预防瘢痕过度生长或瘢痕疙瘩的形成。
Objective properly deal with open wounds to check and prevent scars from overgrowing and the producing of the keloids.
免疫组化染色显示瘢痕疙瘩中成纤维细胞有丰富的I型前胶原表达。
Immunohistochemical staining showed increased expression of new formed, type I procollagen in keloid tissue.
目的探讨结缔组织生长因子在增生性瘢痕和瘢痕疙瘩发病机制中的作用。
Objective to explore the effect of connective tissue growth factor on the pathogenesis of hypertrophic scar and keloid tissue.
硅凝胶薄膜可用于治疗和预防瘢痕疙瘩和增生性疤痕，是用来对愈合的皮肤。
Silicon gel sheeting can be used to treat and prevent hypertrophic and keloid scars and is used on healing skin.
方法：取瘢痕疙瘩和正常皮肤，采用组织块培养法进行成纤维细胞体外培养。
METHODS: Keloid and norm al skin tissue were collected for the fibroblasts in vitro using tissue-block cultivation.
结果：64块瘢痕疙瘩变为线状或萎缩性瘢痕，随诊1 ~6年，均获良好控制。
Results 64 cases of keloids became line-shape or atrophic scar and showed good control in 1 ~ 6 years follow-up.
结论P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞MMP1、MMP3蛋白的表达。
Conclusion P53 can obviously inhibit MMP1 and MMP3 expression in human keloid fibroblasts.
另一方面，也可增厚，变粗糙和如索状，形成肥厚性疤痕或瘢痕疙瘩，引起重度瘙痒。
On the other hand, sometimes they grow thick, tough, and corded, forming a hypertrophic scar or keloid, and may cause severe pruritus.
S100蛋白基因成员在增生性瘢痕和瘢痕疙瘩中的差异表达有助于两者的鉴别诊断。
S100 protein genes differentially express in hyperplastic scar and keloid, which facilitate the differential diagnosis between the two.
S100蛋白基因成员在增生性瘢痕和瘢痕疙瘩中的差异表达有助于两者的鉴别诊断。
S100 protein genes differentially express in hyperplastic scar and keloid, which facilitate the differential diagnosis between the two.
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