当试图向端部运动时,收缩的组织结构被不经意的伸展和破坏,并刺激疼痛受体。
Upon attempting to move to end-range, shortened structures are prematurely stretched and deformed which stimulates nociceptive receptors.
受损细胞向你的大脑发出信息,传递受伤的信号并激活疼痛受体,使得你的皮肤对触碰变得敏感。
Damaged cells send messages to your brain, signaling they're injured and activating pain receptors, which makes your skin sensitive to touch.
AID发射一道不可见的毫米级波长的能量束,其渗透深度正好足够深,让目标皮肤下的疼痛受体受到刺激。
The AID directs an energy beam, which is in the invisible millimeter wavelength, that penetrates just deep enough beneath the skin to make the target's pain receptors shout.
MC1R基因属于包括脑部疼痛受体在内的受体家族,可想而知该基因的变异也会对身体疼痛敏感性产生影响。
The MC1R gene belongs to a family of receptors that include pain receptors in the brain, and as a result, a mutation in the gene appears to influence the body's sensitivity to pain.
在杂志发表的内容中,内德·戈德的研究小组描述了针灸是怎样使三分之二的正常小鼠减缓疼痛,而无法在缺乏腺苷酸受体基因的对照组小鼠身上生效的。
Writing in the journal, Nedergaard's team describe how acupuncture reduced pain by two thirds in normal mice, but had no effect on the discomfort of mice that lacked the adenosine receptor gene.
然后,细胞附近神经上的腺甙受体收集这种物质,并通过减轻疼痛作出反应。
This is then picked up by adenosine receptors on nearby nerves, which react by damping down pain.
因此,能够抑制辣椒素受体的物质,应该就可以减轻由发炎引起的疼痛。
Substances that inhibit the capsaicin receptors should therefore dampen inflammatory pain.
其他药物的目的在于通过阻断或激活TRPV 1受体减少疼痛和炎症,这还在研究中。
Other drugs aimed at reducing pain and inflammation by blocking or activating TRPV1 receptors are in the research pipeline.
我想疼痛的受体可能存在差别。
这些受体可以感受热,炎症触机点并传导疼痛。
These receptors can sense heat, trigger inflammation and transmit pain.
科学家一直知道神经系统受体如已知的TRPV 1能影响机体的疼痛感觉。
Scientists have long known that the nervous system receptor known as TRPV1 can affect sensations of pain in the body.
香草酸受体是瞬时受体电位离子通道家族中的成员,为治疗疼痛的新靶点。
As the member of transient receptor potential family of ion channels, vanilloid receptor is the new target against pain.
目的:通过调节辣椒素受体1 (VR1)基因表达,对VR 1基因的功能及疼痛的基因治疗进行基础研究。
Objective: By regulation of VR1 gene expression, fundamentally investigate function of VR1gene and pains gene therapy.
脑啡肽是机体产生的一种类型的缓解疼痛感觉的阿片受体,但在慢性疼痛的情况下,这些物质似乎流量不够。
Enkephalins are a type of opiate that the body produces to dull pain sensations, but in cases of chronic pain, these agents appear not to flow in sufficient quantities.
我们发现随着异源二聚体的具体,大脑选择性的对吗啡治疗慢性疼痛起作用,阻碍单个受体对吗啡止痛作用的传导。
"We found that the brain selectively responds to chronic morphine by increasing heteromer abundance, blocking individual receptors from signaling the analgesic response to morphine, " said Dr. Devi.
因此,P 2x4受体和细胞内信号传导介质中的小胶质细胞是有希望的治疗靶点,为开发新的药物在神经性疼痛的管理。
Thus, P2X4 receptors and the intracellular signaling mediators in microglia are promising therapeutic targets for the development of novel pharmacological agents in the management of neuropathic pain.
因此,P 2x4受体和细胞内信号传导介质中的小胶质细胞是有希望的治疗靶点,为开发新的药物在神经性疼痛的管理。
Thus, P2X4 receptors and the intracellular signaling mediators in microglia are promising therapeutic targets for the development of novel pharmacological agents in the management of neuropathic pain.
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