结论MMI对体外培养人甲状腺细胞的功能有影响。
Conclusions MMI plays an important role in affecting thyroid cells function in cultured human thyroid follicles.
结果提示:甲状腺细胞膜电位与动物机能状态有密切关系。
These results suggest that the cellular membrane potential of thyroid gland is closely related to the animal functional states.
常规病理学法5%或以上甲状腺细胞染色为阳性,低于5%甲状腺细胞染色为阴性。
Classical pathologic method: cases that showed specific staining of 5% or more of the tumor cells, regardless of staining intensity, were scored as positive for TPO.
补碘及补碘补锌组甲状腺细胞高度、滤泡直径、胶质含量和超微结构与对照组相一致。
The height of epithelium, the diameter of follicle, the contents of glue and the morphology of ultra-structure of thyroid in Zn-supplementation groups were similar to that in control group.
目的研究硒对H_2O_2诱导的体外培养人甲状腺细胞氧化损伤、凋亡及细胞超微结构的影响。
Objective To investigate the influence of selenium(Se) on oxidative damage, apoptosis and ultrastructure of human thyrocyte induced by H_2O_2 culture in vitro.
1912年德国医生开始尝试用健康的甲状腺细胞来医治患有甲状腺功能减退的孩子,但是收效甚微。
In 1912 German doctors attempted to treat children who had underactive thyroids with normal thyroid cells, but to little avail.
甲状腺器官形态形成早期阶段,某些转录因子通过激活甲状腺特异基因的转录,对甲状腺细胞迁移、增殖与分化起重要作用。
Genes involved at early stage of the morphogenesis play important roles in thyroid cell migration, proliferation and differentiation by activating thyroid-specific gene transcription.
结果低浓度的KI对人甲状腺细胞生长没有影响,而中、高浓度的KI对细胞生长具有抑制作用,并且呈现剂量-时间依赖关系;
Results KI of middle and high dose can inhibit human thyrocytes to proliferate and exhibit dose-effect and time-effect relationships.
结果低浓度的KI对人甲状腺细胞生长没有影响,而中、高浓度的KI对细胞生长具有抑制作用,并且呈现剂量-时间依赖关系;
Results KI of middle and high dose can inhibit human thyrocytes to proliferate and exhibit dose-effect and time-effect relationships.
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