结论电子直线加速器照射损伤可诱导细胞凋亡。
Conclusion The electron linear accelerator radiation injury can induce cell apoptosis.
通过你的皮肤可以透视出你的生活，不管是怀孕时脸上的一抹红晕，还是太阳照射损伤产生的淡淡斑点。
Your skin can reveal the stories of your life, from the fabled glow of pregnancy to the less-welcome spots that surface from sun damage.
探讨抗氧化剂 -丁羟甲苯、维生素C和维生素 E 是否对紫外线照射诱发的DNA损伤具有拮抗作用。
Investigate the role of antioxidants-butylated hydroxytoluene, vitamin C and vitamin E in UVR -induced DNA damage.
结论适宜浓度的烟酰胺是抗紫外线照射诱发DNA损伤的拮抗剂。
Conclusion nicotinamide is a perfect antagonist to UVR-induced DNA damage within fitting concentration.
结果表明，在不同照射条件下辐射损伤的程度和特性不一，由辐射所诱发的体细胞效应大多数是可以恢复的。
The results indicated that the degree and characteristic of radiation damage were variable under different irradiation conditions and the somatic cell responses induced by irradiation were reversible.
方法实验动物均选用SPF级C57BL小鼠，用不同药物和不同照射剂量处理后，建立急性放射损伤模型。
Methods to establish acute radiation damage pattern, C57BL mice in SPF grade were divided into different groups by different ways and irradiation doses.
本文研究了大鼠颈部脊髓分割照射后的亚致死性损伤修复过程。
The kinetics of repair of sublethal damage was studied on cervical spinal cord in rats after fractionated irradiation.
表明FMCM对照射造成的造血损伤具有良好的保护和治疗作用。
The above results suggest that FMCM has good protective and promoting effects on reducing hematopoietic damage caused by irradiation.
结论：迁延照射造成的细胞遗传学损伤较一次照射严重，且修复缓慢。
CONCLUTION: There were more severe cytogenetic damage and slower repair in continual exposure to radiation than in a single irradiation.
目的：通过单细胞凝胶电泳技术检测不同强度激光照射对小鼠脾淋巴细胞DNA的损伤程度。
Objective: To detect DNA damage grade after spleen lymphocytes of mouse were irradiated by laser.
慢照射辐射损伤小，突变频率高，有益突变多。
The chronic irradiation was minor in injury and higher in frequencies of beneficial variation.
存活细胞经二次照射后，SCGE结果和微核发生率结果表明，首次照射剂量与子代二次照射后的损伤程度存在剂量效应关系。
After the survival cells were given second irradiation, both results of SCGE and micronucleus frequency showed that the second damage was correlated with the original irradiation doses.
方法通过梭曼的离体透皮吸收、紫外线照射致皮肤损伤和组织切片方法进行这两种皮肤特点的比较。
Methods the ways of skin injury of ultraviolet radiation skin permeation of soman and section of skin tissues were used for the study.
目的探讨紫外线照射自血回输（AUVIB）疗法对梭曼染毒家兔肺损伤的治疗作用。
Objective To study the therapeutic effect of autotransfusion of ultraviolet-irradiated blood(AUVIB) on the pulmonary damage due to soman intoxication in rabbits.
但大剂量照射造成的辐射损伤是影响其性能的最大因素。
However, the radiation damage by high total dose of exposure affects its performance.
结果表明，抗辐射菌经重离子射线照射后所引起的DNA二条链的切断损伤经过培养能被修复；切断的DNA二条链的修复时间随着照射剂量的增加而延长；
The results showed that the DR's DNA double strand break damage caused by heavy ion beams could be repaired through culture, and the repairing time extended as the increment of radiation dose.
结论：强脉冲光照射对V79细胞DNA无损伤。
对照射后蚕豆根尖下细胞中的染色体损伤情况进行了研究。
Studied on chromosome damage in cells of broad bean root tip.
激光损伤程度与照射脉冲次数相关。
结论：冷冻联合激光照射对兔耳静脉有较强的损伤作用，具有治疗静脉畸形的可能性。
Conclusion: Liquid nitrogen cryotherapy combined with Nd:YAG laser radiation could result in major injury to the auricular veins of rabbits and may be applied for venous malformation treatment.
结论：视神经间接损伤后，早期适量的X射线局部照射能有效地抑制胶质细胞的反应性增生，能明显地促进神经轴突的结构再生。
It is suggested that early proper X-ray irradiation on the injured optic nerve can effectively inhibit the proliferation of astrocytes and obviously promote the axonal regeneration of optic nerve.
目的应用已制备的兔肝硬化肝癌模型观察肝病状态下肝癌照射后放射性肝损伤的细胞因子的变化。
Objective To observe the changes of the cytokines following stereotactic irradiation for hepatocarcinoma with cirrhosis in rabbits.
目的探讨X线照射能否促进大鼠压迫型脊髓损伤区结构及损伤后功能的恢复。
To explore whether X-irradiation can enhance the functional and structural recovery of the injured spinal cord of rats.
结论：在光线照射后24h之内，视网膜损伤是一个逐渐增强的过程。
Conclusions: the damage of the retina and choroid became stronger during the first 24 hour after the irradiation.
经过同样剂量紫外线照射，基质对照组皮肤外观及各层皮组织结构均可见显著改变。结论：研究初步表明纳米蒙脱土具有抗紫外线所致皮肤损伤特性。
Results: The skin tissue in the montmorillonite intervene group irradiated by ultraviolet rays didn' t change obviously as compared with that in the self-control group. But, the damage skint…
目的探讨烟酰胺对紫外线照射诱发DNA损伤的保护作用。
Objective To investigate the protective effect of nicotinamide on UVR-induced DNA damage.
结果表明UVA照射后1o2和O2 -。的水平与由此引发的脂质过氧化损伤存在正相关关系。
Experiment results indicate that UVA induced lipid oxidative damage has a positive relationship with 1o2 and O-2 · level.
然而，胸壁照射不可避免会照射一定体积的心肺组织，从而引发心肺损伤。
However, chest wall irradiation can also affect the heart and lungs by exposing some of them to the radial and do harm to them.
细胞化学分析证明，受激光照射的染色体区段的DNA遭到了损伤。
Cytochemical analysis demonstrated that chromosomal DNA at the specific irradiated region was impaired.
细胞化学分析证明，受激光照射的染色体区段的DNA遭到了损伤。
Cytochemical analysis demonstrated that chromosomal DNA at the specific irradiated region was impaired.
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