该过程是细胞应答反应之一，它会激活产生炎症的蛋白质，甚至导致细胞死亡。
This process is one of many cellular responses that activate proteins that increase inflammation and can even result in the death of the cell.
结论：RANTES参与了哮喘的发病过程，其机制很可能与它能够激活炎症细胞有关。
Conclusions: RANTES is involved in the asthmatic process possibly by activating inflammatory cells.
结论体外循环可诱导炎症反应致内皮细胞损伤或激活，并可致血管通透性增高。
Conclusion CPB can induce activation damage of vascular endothelial cell, and increase vascular permeability.
目的研究前炎症细胞因子对人胸膜间皮细胞（HPMC）释放纤溶酶原激活物抑制剂（PAI1）的诱导效应。
Objective To investigate effects of two proinflammatory cytokines on the production of plasminogen activator inhibitor 1(PAI 1) by human pleural mesothelial cells(HPMC).
目的探讨白细胞激活及炎症反应在慢性静脉功能不全中的作用。
Objective To discuss the role of leukocyte activation and inflammatory processes in the disease of chronic venous insufficiency (CVI).
在炎症过程中，细胞被激活，同时数种炎症介质被释放包括。
During an inflammatory process cells become activated and several mediators including tissue factor (TF) are released.
牙周优势菌lps作用于单核巨噬细胞，诱导产生各种细胞因子和炎性介质，参与牙周病的炎症激活和组织创伤。
LPS involves in the inflammatory process and tissue destruction during periodontal diseases through inducing mononuclear macrophages to produce various cell factors and inflammatory mediators.
各种途径的巨噬细胞激活后引起细胞对炎症和纤维化的不同应答。
Different pathways of macrophage activation result in cells with widely diverse effects on inflammation and fibrosis.
研究发现过敏炎症中有许多细胞因子和趋化因子参与，抗组胺药物对炎症细胞的迁移和激活有抑制作用。
It has been reported that a lot of cytokines and chemokines were involved in allergic inflammation. Antihistamines have inhibition effects on the migration and activation of inflammatory cells.
弗斯可林的其他功效同样显著，包括抑制象由血小板激活因子(PAF)6引起的炎症以及抑制癌细胞的扩散。
Forskolin has other reported USES as well, including inhibition of the pro-inflammatory substance known as platelet-activating factor (PAF) 6 and inhibition of the spread of cancer cells.
PTCA后白细胞的激活可能触发一系列病理生理联级反应，释放某些炎症介质，促进血管平滑肌增殖导致再狭窄；
Activation of leukocytes after PTCA can trigger a serial cascade of pathophysiologic reaction, releasing some inflammation intermediates lead to restenosis.
降低 低水平的 炎症核内皮细胞激活可能是酒精饮料发挥心血管保护作用的另一个可能机制。
Reducing low-grade inflammation and endothelial activation may be another potential mechanism by which alcoholic beverages exert their cardioprotective effect.
降低 低水平的 炎症核内皮细胞激活可能是酒精饮料发挥心血管保护作用的另一个可能机制。
Reducing low-grade inflammation and endothelial activation may be another potential mechanism by which alcoholic beverages exert their cardioprotective effect.
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