近年来炎症学说在胰岛素抵抗及2型糖尿病发病机制中的作用备受关注。
Inflammation plays an important role in the pathogenesis of insulin resistance and type 2 diabetes.
炎症学说的提出为糖尿病的发病机制和开发预防糖尿病新药提供了又一个新的研究方向。
Inflammation hypothesis of diabetes will provide a new research field in the pathogenesis of diabetes and in the development of new preventive and therapeutic drugs for diabetes.
除免疫性炎症学说外，神经源性炎症学说越来越受到关注，但对神经系统与这些炎症过程复杂的相互作用了解较少。
In addition to the theory "immuno-inflammation", the theory "neurogenic inflammation" is arousing the interest of scholars more and more.
前言：动脉粥样硬化发病机制的“炎症-损伤-反应”学说得到广泛支持和认可。
The theory, believing that inflammation-injury-reaction makes the pathogenesis of atherosclerosis, has been widely accepted.
本文总结了体重减轻、胃肠激素、前肠学说、后肠学说、脂肪细胞因子和炎症因子等方面机制的研究进展。
This review summarizes the possible mechanisms which include weight loss, gastrointestinal hormones, foregut hypothesis, hindgut hypothesis, adipocytokines, and inflammatory factors.
本文总结了体重减轻、胃肠激素、前肠学说、后肠学说、脂肪细胞因子和炎症因子等方面机制的研究进展。
This review summarizes the possible mechanisms which include weight loss, gastrointestinal hormones, foregut hypothesis, hindgut hypothesis, adipocytokines, and inflammatory factors.
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