炎症介质可能在A型行为高血压患者早期肾脏损害中起重要作用。
Inflammatory mediators perhaps plays role in early renal damage in patients with type a behavioral hypertension.
其中细胞因子和炎症介质起着重要作用。
The cytokines and mediators of inflammation played an important role.
皮肤损伤愈合是机体保持组织完整性的一个有组织的过程，需要炎症细胞、生物化学介质之间的复杂作用。
Skin wound healing is an organized process for keeping cutaneous integrity, which needs the complicated interaction between inflammatory cells and biochemical mediators.
目的探讨炎症介质在高血压肾功能损害中的作用。
Objective to study the influence of inflammatory mediators on renal damage in patients with essential hypertension (EH).
目的探讨低剂量环磷酰胺对烧伤大鼠全身炎症介质综合征（SIRS）的控制作用。
Objective To study the effect of low dose cyclophosphamide on SIRS (systemic inflammatory response syndrome)in early stage of burns.
本文主要针对增生性瘢痕的形成机制，从病理生理、炎症介质、局部细胞因子的相互作用和物理环境产生的影响做一综述。
This review will examine the mechanisms of hypertrophic scar formation, from the aspects of the pathophysiology, inflammatory mediators, the action of local cytokine and the influence on physical...
阿托伐他汀可抑制炎症介质的分泌及表达，具有抗炎作用。
Atrovastatin suppress expression of inflammatory mediator. It has an effect of anti-inflammation.
AR与BA的发病机制至今仍不十分清楚，其是涉及多种炎性细胞的相互作用，诸多炎性介质、细胞因子、转录因子等共同参与的慢性炎症反应。
The pathogenesis of AR and BA has not been quite clearly, it may be involved in the interaction of many inflammatory mediators, cytokines and transcriptional factors.
牙周优势菌lps作用于单核巨噬细胞，诱导产生各种细胞因子和炎性介质，参与牙周病的炎症激活和组织创伤。
LPS involves in the inflammatory process and tissue destruction during periodontal diseases through inducing mononuclear macrophages to produce various cell factors and inflammatory mediators.
结论：氧化苦参碱可在一定程度阻断LPS对PDLC的破坏，抑制牙周炎症介质的表达，对PDLC有保护作用。
Conclusion: Oxymatrine can interrupt LPS's damage to PDLC in some extent, prohibit the delivery of inflammation medium, and provide protection to PDLC.
鉴定象IL-23一样的特异性炎症介质打开了新的靶向抗炎治疗的大门，并大大减少副作用。
The identification of specific inflammatory mediators like IL-23 opens the door to developing targeted anti-inflammatory treatments which may have fewer side effects.
炎症时释放的许多炎症介质都能够与TRPV 1发生相互作用，产生疼痛或痛觉过敏，并且通过各种不同的信号通路来调制TRPV1的活性。
In inflammatory pain, a number of inflammatory mediators are released, which modulate TRPV1 activity through various signal pathways or interact with TRPV1 generating pain or hyperalgesia.
结论榜嘎总生物碱具有一定的抗炎作用，其机制包括抑制多种炎症介质。
Results The total alkaloids on Herba Aconiti inhibited the auricular edema, decreased the permeability…
结论榜嘎总生物碱具有一定的抗炎作用，其机制包括抑制多种炎症介质。
Results The total alkaloids on Herba Aconiti inhibited the auricular edema, decreased the permeability…
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