目的研究激活诱导细胞死亡（AICD）现象在乙型肝炎慢性化和重型化机制中的意义。
ObjectiveTo study the significance of AICD(activation-induced cell death) in the pathogenesis of chronic hepatitis B and chronic severe hepatitis B.
活性氧可以诱导细胞程序性死亡或坏死、可以诱导或抑制许多基因的表达，也可以激活上述级联信号。
Ros induce programmed cell death or necrosis, induce or suppress the expression of many genes, and activate cell signalling cascades, such as those involving.
活性氧可以诱导细胞程序性死亡或坏死、可以诱导或抑制许多基因的表达，也可以激活上述级联信号。
Ros induce programmed cell death or necrosis, induce or suppress the expression of many genes, and activate cell signalling cascades, such as those involving.
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