皮肤或血液的检查可以发现潜伏感染。
潜伏感染是指病毒在大部分时间是隐伏的。
They are those in which the virus persists in an occult, or cryptic, form most of the time.
TK基因对病毒嗜神经性，缺失后有抗潜伏感染的特性。
TK gene represent philic-nerves and its deletion enhances the ability of anti-latent infection.
PRV的潜伏感染及与其他病毒的混合感染问题值得注意。
It is worth to set eyes on the problem of PRV latency infection and infect with other virus.
能在神经系统建立潜伏感染是嗜神经性疱疹病的一个主要特征。
That latency infection established in the nervous system was a main character of hydropholibic herpesvirus.
另外，HPV潜伏感染目前临床上尚无有效的诊断方法，而且是复发的主要原因。
In addition, the latent infection of HPV that is main reason of recurrence has not effective method to diagnose clinically now.
除心理因素外，尖锐湿疣复发还有下面这些原因：①亚临床感染及潜伏感染的存在。
In addition to psychological factors, there are the following reasons: the presence of subclinical infection and latent infection.
大多数情况下都是潜伏感染，即患者虽然受到感染，但没有症状，不太可能传染给他人。
Most cases are latent, meaning patients are infected but have no symptoms and are very unlikely to infect anyone else.
成功构建了HCMV先天潜伏感染再激活小鼠小肠炎模型，病理表现为出血性坏死性肠炎。
A rat model of enteritis induced by reactivation of congenital latent HCMV infection was successfully established, and its pathological presentations indicate hemorrhagic necrotic enteritis.
Ustekinumab是一种选择性的免疫抑制剂，可能有可能增加感染的危险，并重新恢复潜伏感染。
Ustekinumab is a selective immunosuppressant and may have the potential to increase the risk of infections and reactivate latent infections.
因此，细菌生长较慢且杀死小鼠的可能性减少。将来的研究能够调查这些潜伏感染是否能防御其它病毒感染。
As a result, the bacteria grew more slowly and were less likely to kill the mice. Future research can investigate whether these latent infections protect against other viruses.
局部的讨论根除是生物体hiv潜伏感染后的模型，具有多种药理作用的研究方法延迟刺激原病毒基因的表达。
Topical to the discussion of HIV eradication is the biology behind latent infection as well as models for the study of latency and pharmacological approaches to stimulate proviral gene expression.
干扰素刺激基因所编码干扰素刺激蛋白的高水平表达可以抑制病毒的复制，这种抑制是导致慢病毒建立潜伏感染的原因之一。
The over expression of interferon stimulated protein can inhibit the replication of viruses. The inhibition can lend lentivirus to establish latent infection.
本研究以牛免疫缺陷病毒（BIV）为慢病毒模型，对一种干扰素刺激蛋白ISG15在BIV潜伏感染中的作用进行了初步的探讨。
In this work, we used BIV as a lentivirus model, and did a preliminary study on the function of bovine ISG15 (bISG15) in latent infection of BIV.
外界刺激、宿主免疫力的降低等因素都可以使宿主由潜伏感染状态发病，成为带毒者和传播源，因此，对潜伏感染建立及维持机理的研究就显得尤为重要。
Hosts can be diseased from latency condition by the decreasing of host's immunity and stimulation of condition, and so on , then become a carrier and infectious source.
其中维持最长时间的治疗方法是医生怀疑是由于持续的，潜伏的细菌感染，并由此开出长期或者无限期的抗生素药方。
For the longest time, doctors suspected that the condition was caused by persistent, hidden bacterial infections and sometimes prescribed long-term or open-ended antibiotic therapy.
动物实验的成功促使研究小组进行试管研究，以确定二甲胺四环素疗法能否影响感染HIV的人类T细胞的潜伏病毒。
The success with the animal model prompted the team to study in test tubes whether minocycline treatment affected latency in human t cells infected with HIV.
一个人一旦染上恰加斯病，这种感染可能潜伏数十年。
Once a person has contracted Chagas disease, the infection may remain dormant for decades.
成虫的游走和钻出(潜伏期)在感染后的10至14个月之间出现。
The migration and emergence (incubation period) of the worm (s) take between 10 to 14 months after infection.
对密切接触者进行了跟踪，在两倍于禽流感感染潜伏期期间未发现症状。
Contacts have been followed up and have shown no symptoms at twice the incubation period for avian influenza infection.
几乎所有肺结核病菌感染都有潜伏期，携带者没有症状，而且没有传染性。
Nearly all TB infections are latent, with carriers showing no symptoms and they are not infectious.
全世界每三个人中就有一人感染潜伏性结核病菌(即结核细菌)。
One in three people in the world is infected with dormant TB germs (i.e. TB bacteria).
最后一位病毒感染患者于2009年1月1日死亡，这已超过了埃博拉最长潜伏期(42天)的两倍以上。
The last person to be infected by the virus died on 1 January 2009. This is more than double the maximum incubation period (42 days) for Ebola.
感染后的潜伏期大概会持续10至14天。
The incubation period lasts approximately 10-14 days after contact.
感染后的潜伏期大概会持续10至14天。
The incubation period lasts approximately 10-14 days after contact.
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