表明过量氟对滑膜细胞有损伤作用。
These suggest the excessive fluoride may bring about damage to the joint synovial cells.
结果：成功体外培养人关节滑膜细胞。
滑膜细胞形成被称为滑膜衬里层的特殊结构。
The Synoviocytes form a distinct structure called the synovial lining layer.
结果青藤碱组CIA大鼠滑膜细胞的凋亡数明显增加。
Results Sinomenine could induce apoptosis of synoviocytes in CIA rats.
方法原位细胞凋亡检测试剂盒检测各组原代大鼠滑膜细胞凋亡的水平。
Methods the level of the apoptosis in different groups were assayed by the in situ cell death detection kit.
结论通痹灵治疗类风湿性关节炎的机制可能与诱导滑膜细胞凋亡有关。
Conclusion the mechanism of curative effect of Tongbiling on arthritis concern with rising the level of apoptosis.
结论青藤碱治疗类风湿性关节炎的机制可能与诱导滑膜细胞凋亡有关。
Conclusi on the mechanism of curative effect of sinomenine on arthritis concern with rising the level of apoptosis.
结果随着羧甲基几丁糖浓度的增加，类风湿性关节炎模型滑膜细胞的数量减少。
Results the number of the adjuvant arthritis synovial cells were decreased as the concentration of O-Carboxymethyl Chitosan increased.
方法行关节腔镜手术取RA患者膝关节滑膜组织消化分离滑膜细胞进行分组培养。
Method Synovial cells from the knee joint in patient with RA were digested, divided and separately cultured after arthroscopy.
滑膜细胞培养基是专门为正常人类滑膜细胞体外培养设计的最适于其生长的培养基。
Synoviocyte Medium (SM) is a complete medium designed for optimal growth of normal human synoviocytes in vitro.
目的：探讨佐剂性关节炎(aa)大鼠滑膜细胞的类肿瘤样增生和相关基因表达的机制。
AIM: To investigate the mechanism of the tumor-like proliferation of synoviocytes in adjuvant arthritis (AA) rats.
其主要病理特点是滑膜细胞增生、衬里层增厚、多种炎性细胞浸润、血管翳形成，以及软骨和骨组织的破坏。
Histopathologic features of RA encompass infiltration by macrophages and T cells synovial lining hyperplasia, neoangiogenesis, pannus formation and destruction of cartilage and bone.
目的：建立佐剂性关节炎(aa)大鼠成纤维样滑膜细胞(FLS)的体外培养及鉴定方法，为进一步的实验研究奠定基础。
Objective: To establish the methods of culture and identification of fibroblast-like synoviocytes (FLS) in rats with adjuvant arthritis (AA) in vitro, and provide a base for further study.
方法：检测大鼠非致炎侧足爪体积，血清丙二醛(MDA)，滑膜细胞产生白细胞介素1 (IL 1)和亚硝酸盐(NO-2)量。
Methods: The volume of non-injected hind paw of AA rats, serum malondialdehyde (MDA) content, interleukin-1 (IL-1) and nitrite (no - 2) produced from articular synoviocytes were measured.
组织，细胞质蛋白，人肿瘤，滑膜肉瘤。
滑膜组织内出现散在出血点及炎细胞浸润，滑膜表面完整性破坏。
There were many hemorrhagic spots in synovium with infiltration of inflammation cells and the surface of synovium was destructed.
目的研究羧甲基几丁糖对兔膝关节类风湿性关节炎模型滑膜组织细胞体外培养的影响。
Objective to study the effects of the O-Carboxymethyl Chitosan on the cultivation of synovial sells from Rabbits adjuvant arthritis in vitro.
可改善滑膜淋巴细胞浸润情况。
因此，和在这个模型中的应用一样，我们不推荐滑膜内屈肌腱修复应用碱性成纤维细胞生长因子。
Therefore, as applied in this model, basic fibroblast growth factor is not recommended for intrasynovial flexor tendon repair.
其细胞形态与滑膜被复细胞相似且兼有A、B型细胞的特点，并有类分泌现象。
Cytologically, the tumor cells appear similar to the synovial lining cells and maintain the characteristics of both A-cells and B-cells and appear to have secretory function.
结果：滑膜下致密纤维组织中弥漫浸润的巨噬细胞和异物巨细胞内有大量磨屑。
Results: in the dense fibrous connective tissues, there are infiltration of macrophages and foreign-body giant cells with plenty of wear debris.
结果：12例低分化滑膜肉瘤中小细胞型4例，大细胞型6例，高度恶性梭形细胞型2例。
Results in 12 cases of poorly differentiated synovial sarcoma, 4 were small cell type, 6 were large cell type and 2 were high grade spindle cell type.
目的通过观察激素治疗兔关节炎模型过程中滑膜组织内血管内皮细胞生长因子(VEGF)的基因表达，进一步探讨激素治疗关节炎的作用机制。
Objective to observe the expression of vascular endothelial growth factor (VEGF) in synovial tissue during treatment of rabbit arthritis model with corticosteroids and explore its mechanism.
结果与模型对照组比较，尪痹片能够显著抑制炎症细胞的浸润和滑膜增生，减轻软骨损伤程度（P<0.05）。
Results WP remarkably inhibited the hyperplasia of synovial membrane and the infiltration of inflammatory cells, and reduced cartilage injury as compared with the model group(P < 0.05).
腱鞘巨细胞瘤可发生于关节滑膜、滑囊或腱鞘，依据其生长方式和生物学行为，可分成局限性和弥漫性腱鞘巨细胞瘤两类。
Tenosynovial giant cell tumors arise from synovium of joints, bursae, or tendon sheaths, and are classified into localized and diffuse types based on the growth pattern and clinical behavior.
其结果大多数癌除肝细胞肝癌、肾透明细胞癌外，大部分肿瘤细胞显示中等度到强阳性。在滑膜肉瘤、恶性间皮瘤及胸腺瘤上皮样成份弱阳性。
The majority of the carcinomas, except hepatocellular, adrenocortical clear cell ones are stained of strong or moderate intensity in the majority of tumor cells.
关节炎高峰期见三足步态，组织严重水肿，关节腔明显积液；关节液、关节滑膜及其周围组织见大量炎性细胞浸润，较好地模拟了人急性痛风性关节炎的病变特点。
At the peak of arthritis, the rats showed tri-legged gait, and severe edema in the tissue, as well as hydrarthrosis with massive leukocytes infiltrating into joint cavity and the tissue around.
模型组关节液白细胞计数显著高于对照组，病理学观察有急性炎症发生，表现为表面滑膜组织肿胀、坏死、结缔组织水肿、炎症细胞弥漫性浸润等；
AA was also found by pathological observation, with swelling, necrosis in the surface synovial tissues. Edema and diffusive infiltration of inflammatory cells happening in connective tissue as well.
结论：滑膜巨噬细胞在AA大鼠关节破坏中起重要作用。
CONCLUSION: Synovium macrophages of AA rats may play an important role in articular damage.
目的：了解佐剂性关节炎(aa)大鼠滑膜巨噬细胞在关节破坏中的作用。
AIM: To explore the role of synovium macrophage of adjuvant arthritis (AA) rats in articular damage.
应用推荐