不过，披衣菌却有办法让进入细胞的囊泡避开溶小体，使披衣菌与受感染细胞的其他部份隔离，而能自由增殖。
But chlamydiae somehow compel their entry vacuoles to avoid lysosomes, enabling the bacteria to proliferate freely while separated physically from the rest of the infected cell.
而如果共同感染的病毒数目超过一个特定的阈值，则cl基因主导的正反馈环路将使细胞转向溶原通路。
If the number of co-infecting viruses exceeded a certain threshold, the positive feedback loop associated with cI dominated, turning the switch to the lysogenic pathway.
研究者还发现2型纤维蛋白溶酶原激活因子抑制物(PAI2)(一种丝氨酸蛋白酶抑制物)抑制结核杆菌感染的巨噬细胞内的膜联蛋白- 1的蛋白水解作用。
The researchers also found that plasminogen activator inhibitor type 2 (PAI2), a serine-protease inhibitor, inhibited proteolytic truncation of annexin-1 in macrophages infected with M. tuberculosis.
研究者还发现2型纤维蛋白溶酶原激活因子抑制物(PAI2)(一种丝氨酸蛋白酶抑制物)抑制结核杆菌感染的巨噬细胞内的膜联蛋白- 1的蛋白水解作用。
The researchers also found that plasminogen activator inhibitor type 2 (PAI2), a serine-protease inhibitor, inhibited proteolytic truncation of annexin-1 in macrophages infected with M. tuberculosis.
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