尼氏染色对海马神经元进行半定量计数分析。
Semiquantitative analysis of hippocampal neurons was carried out by using Nissl stain.
结果发现SVHRP可致海马神经元兴奋性降低。
The results showed that SVHRP could decrease the excitability of hippocampal neurons.
完整的海马神经元其形态至少可维持4小时以上。
The integrated hippocampal neurons can live 4 hours at least in the standard bath solution.
目的观察兴奋和抑制海马神经元对慢波睡眠的影响。
Objective To investigate the effects of excitement and inhibition of hippocampal neurons on sleep and wakefulness.
目的探讨褪黑素对海马神经元保护作用的可能机制。
Objective To study the protective mechanism of melatonin on neurons in hippocampus.
病理图文分析系统检测大脑皮质及海马神经元密度。
Neure density in cerebral cortex and hippocampi was analyzed with pathological graphics context analysis system.
目的：探讨半脑照射后大鼠的海马神经元密度变化规律。
Aim:To study the regulation of early changes of neuron density in hippocampus after hemispheic irradiation.
目的建立体外培养大鼠皮质及海马神经元缺氧缺糖模型。
Objective To establish oxygen-glucose deprivation model of rats′ cortex and hippocampusal neurons in vitro.
目的探讨海马神经元癫痫样放电引起神经元丢失的机制。
Objective To explore the mechanism of hippocampal neuronal loss induced by epileptic discharge.
目的：研究体外培养海马神经元不同时期的形态学变化。
Objective: To study the alterations in morphology of cultured hippocampal neurons during different developing stages in vitro.
结果和结论：大鼠脑缺血时间与海马神经元损伤程度密切相关。
Results and Conclusion: Cerebral ischemia time in rats are closely correlated with hippocampus neuron damage degree.
结果表明体外培养的胎鼠海马神经元的网络活动具有温度敏感性。
These showed that the activity of hippocampal neuronal networks is sensitive to the temperature.
方法在培养的大鼠海马神经元上用膜片钳全细胞记录钾通道电流。
METHODS Voltage gated potassium outward current was recorded using whole cell patch clamp techniques in the cultured rat hippocampal neurons.
目的：探讨吡咯喹啉醌（PQQ）对培养海马神经元的促生长作用。
Objective: To investigate the effect of pyrroloquiniline quinone(PQQ) on the growth of hippocampal neurons cultured in vitro.
目的研究氟对原代培养大鼠海马神经元氧化应激和DNA损伤的影响。
Objective To explore the effects of fluoride on oxidative stress and DNA damage in primary hippocampal neurons of rats.
方法：膜片钳全细胞记录测量原代培养大鼠海马神经元膜的钾离子电流。
Methods: The potassium currents of rat hippocampal neurons in primary culture were measured with patch clamp whole cell recording.
目的探讨戊四氮（PTZ）反复点燃对发育鼠海马神经元发生的影响。
Objective To investigate the influence of pentetrazole(PTZ) repeated kindling on hippocampal neurogenesis in developing rats.
目的研究低频脉冲磁场对脑缺血再灌注损伤大鼠海马神经元的保护作用。
Objective To investigate the protective effects of low-frequency pulsed electromagnetic field (LFPMF) on neuron following cerebral ischemia-reperfusion injury in rats.
结果DHA可促进新生大鼠海马神经元的生长发育，促进婴儿大脑的发育。
Results For newborn neurons in the hippocampus of rats growth and development, DHA promoted the development of infant brain.
目的探讨睫状神经营养因子(CNTF)对应激大鼠行为和海马神经元的影响。
Objective to discuss the effect of ciliary neurotrophic factors (CNTF) on behavior and hippocampus neuron of stress rats.
目的研究抑郁症模型鼠海马神经元细胞的凋亡，探讨海马损害的病理生理机制。
Objective to study the pathophysiological mechanism about the apoptosis of neurons in the rat hippocampus of depression model.
结论MK801和尼非地平预处理可以部分地阻断EMP所致的海马神经元损伤；
Conclusion Pretreatment with MK801 and nifedipine can partly block EMP-induced injury of hippocampal neurons in vitro.
目的探讨不同强度的推拉动作所致的推拉效应对大鼠海马神经元损伤的程度和时相性。
Objective To explore the degree of injury and phase of push-pull effect induced by different degrees of push-pull maneuver on hippocampus neurons in rats.
目的探讨内毒素预处理对大鼠前脑缺血再灌注后海马神经元的保护效应及其可能机制。
Objective To investigate the protective effect of endotoxin pretreatment on hippocampal neurons in rat forebrain following ischemia reperfusion and its possible mechanism.
目的探讨电磁脉冲(EMP)致体外培养的海马神经元损伤的机制及药物防护的可能性。
Objective To investigate the mechanism of electromagnetic pulse (EMP) -induced injury of hippocampal neurons in vitro as well as the possibility of protection by medicine.
结论眼针疗法可改善实验性血管性痴呆大鼠学习记忆障碍及海马神经元超微结构的变化。
Conclusion Eye acupuncture can produce an improving effect on learning disorder and dysmnesia, and changes in the ultrastructure of hippocampus neurons in experimental vascular dementia rats.
临床上多种疾病可导致脑缺血，而海马神经元的易损性常被当作研究脑缺血的主要模型。
ObjectiveMany disease can lead to the cerebral ischemia, and the vulnerable hippocampus is often treated as the important cerebral ischemia model.
采用DAPI染色技术观察hpk1反义寡核苷酸对OGD诱导的海马神经元凋亡的作用。
DAPI staining was used to investigate the role of HPK1 antisense oligodeoxynucleotides in hippocampus neuron death induced by OGD.
采用DAPI染色技术观察hpk1反义寡核苷酸对OGD诱导的海马神经元凋亡的作用。
DAPI staining was used to investigate the role of HPK1 antisense oligodeoxynucleotides in hippocampus neuron death induced by OGD.
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