因此，我们也许会感到意外，维生素c这个广为人知的抗氧化剂竟有“助氧化剂(Pro - oxidant)”一面，它可以通过增加活性氧的生成让动脉受惠。
It might therefore come as a surprise to learn that vitamin c, a well-known antioxidant, has a "pro-oxidant" alter ego that can benefit arteries by increasing the production of ROS.
暴露在紫外线辐射下的无保护的皮肤会产生叫活性氧簇(ROS)的有害化合物。
Unprotected skin exposed(12) to UV radiation generates(13) harmful compounds(14) called reactive oxygen species(15) or ROS.
汉森的研究小组发现当皮肤处在紫外线辐射之下，三种广泛用于防晒产品中的紫外线过滤成分也会生成活性氧簇ros。
Hanson's research team found that three UV filters widely used in sunscreens generate ROS when skin is exposed to UV radiation.
但毫无疑问，过量生成活性氧会有害处。
But there's no doubting that producing ROS in excess can be harmful.
实验结果表明，香烟主流烟雾中活性氧的含量与烟草品种及制作工艺存在密切的联系。
The results show that the amount of ROS is dependent on the types of tobacco and cigarette production technology.
这些游离的氧原子能维持它们自己的形式或者和氢原子联合成活性氧(ROS)，ROS能被酶和蛋白分解可逆性影响细胞功能。
These rogue oxygen atoms can remain on their own or combine with hydrogen atoms to form reactive oxygen species (ROS), which wreak havoc with enzymes and proteins and adversely affect cell function.
氧化低密度脂蛋白诱导通过细胞内活性氧簇和脂质过氧化反应产物水平测量的氧化应激。
OxLDL induced oxidative stress, measured by the intracellular levels of reactive oxygen species (ROS) and lipid peroxidation products.
然而，研究人员发现在30摄氏度这种清除过程并不能清除活性氧或阻止氧化损伤的发生。
However, at 30 degrees centigrade the researchers found that the cleanup processes cannot cope with ROS and oxidative damage occurs.
结果表明，不同器官组织中活性氧与抗超氧阴离子自由基含量各异。
Results showed that content of ROS and ASAFR vary between different organs and tissues.
UCP2可调节脂肪酸氧化，控制活性氧簇的产生，抑制胰岛素分泌。
UCP2 limits production of reactive oxygen species (ROS) and inhibits insulin secretion and regulates fatty acid oxidation.
抗氧化剂，包括天然的或合成的化合物，例如某些维生素和矿物质，都能有助于减少由活性氧族带来的危害。
Antioxidants include natural and synthetic chemicals, including certain vitamins and minerals, which help to reduce the damage caused by ROS.
细胞周期进程由多种外界因素调控，活性氧可能在细胞周期进程中发挥重要作用。
Cell cycle progression is regulated by a wide variety of external factors, in which reactive oxygen species (ROS) may play an important role.
线粒体呼吸链电子漏是运动性内源活性氧生成的重要机制之一。
The mitochondrial electron leak is an important source of endogenous ROS in exercise.
目的观察氟诱导的大鼠原代培养海马细胞氧化应激损伤和凋亡及活性氧在细胞凋亡中的作用。
Objective to investigate the oxidative stress and apoptosis induced by fluoride in primary rat hippocampal neurons, and to explore the involved role of reactive oxygen species (ROS) on apoptosis.
该文对于近年来活性氧在气孔运动过程中发挥的作用方面的研究进展进行了综述。
In this paper, research progress about the role of ROS in stomatal movement was reviewed.
许多研究已经证明，机体的正常代谢可以产生活性氧自由基，它有很强的氧化性，是机体产生疾病和衰老的关键之一。
Many studies have shown that the normal metabolism of the body can produce reactive oxygen species (ROS), which has strong oxidation, is the key of human diseases and aging.
我们假定活性氧簇分子参与了脂多糖诱导的肝细胞脂肪堆积。
We hypothesize that reactive oxygen species (ROS) mediate lipopolysaccharide (LPS) -evoked hepatic lipid accumulation.
结果与结论：线粒体在态4呼吸状态下，DCF的荧光强度随时间呈线性增加，表明活性氧以恒定速率产生。
Results and Conclusion: DCF fluorescence showed linear increase in State 4 mitochondria, which suggest ROS produced at constant rate.
大多数压力会直接或间接引起粟酒裂殖酵母的活性氧压力。
A majority of the stresses can directly or indirectly induce the reactive oxygen species (ROS) of S. pombe.
近年来的研究结果表明，氟中毒所致的细胞凋亡是多因素作用的结果，其中G蛋白和活性氧(ROS)是最重要的两个因素。
Recent studies have indicated that fluoride induced apoptosis results from multiple factors, of which G protein and ROS are two important factors.
结果①高原力竭运动使总抗氧化能力、活性氧和丙二醛含量增高（P<0.01），超氧化物歧化酶无统计学意义（P>0.05）。
RESULTS:T AOC, ROS activity and MDA level after altitude exhaustion exercise elevated (P< 0.01), but there was no statistical significance in the SOD activity (P >0.05).
本文介绍了水环境中活性氧的来源、种类和测定。
This paper described the sources, species and measurement of ROS.
目的探讨亚硒酸钠诱导NB 4细胞凋亡过程中，活性氧引起的线粒体膜电位丧失及凋亡作用的相关机制。
Objective to investigate the role of reactive oxygen species (ROS) and ROS-caused mitochondrial transmembrane potential loss in sodium selenite-induced apoptosis in NB4 cells.
活性氧簇(ROS)是神经系统脱髓鞘及血脑屏障破坏的媒介。
Reactive oxygen species (ROS) are mediators of demyelination and disruption of the blood-brain barrier (BBB).
结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生，降低气道炎症和气道高反应性，这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
活性氧可以诱导细胞程序性死亡或坏死、可以诱导或抑制许多基因的表达，也可以激活上述级联信号。
Ros induce programmed cell death or necrosis, induce or suppress the expression of many genes, and activate cell signalling cascades, such as those involving.
确切证据表明，活性氧(ROS)参与了淋巴细胞内一系列生理或病理活动的调节。
Convincing evidence has been provided that reactive oxygen species (ROS) can deliver intracellular signals in lymphocyte either in physiological or pathological status.
确切证据表明，活性氧(ROS)参与了淋巴细胞内一系列生理或病理活动的调节。
Convincing evidence has been provided that reactive oxygen species (ROS) can deliver intracellular signals in lymphocyte either in physiological or pathological status.
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