目的探讨阿托伐他汀对血管平滑肌细胞增殖的影响及对血红素氧化酶1 (HO - 1)表达的作用。
Objective to study the effect of atorvastatin on the proliferation of vascular smooth muscle cells (VSMC) and the expression of heme oxygenase-1 (HO-1).
目的：研究血红素氧化酶1的诱导剂高铁血红素在对抗大鼠心肌缺血-复灌损伤中的作用及其相应机制。
Objective: To investigate the effects of heme oxygenase 1 inducer hemin on protection of ischemia-reperfusion injury in rats and its mechanisms.
内源性一氧化碳是一种气体信使分子，是血红素的降解产物。
Endogenous carbon monoxide is a gaseous messenger, which is the degration product of heme.
目的探讨慢性肺心病肺动脉高压患者中血红素氧合酶(HO) - 1和内源性一氧化碳(CO)的变化。
Objective To study the changing of heme-oxygenase-1 (HO-1) and endogenous carbon-monoxide (HbCO) in patients with chronic pulmonary hypertension.
内源性一氧化碳是一种气体信使分子，是血红素的降解产物。
Endogenous carbon monoxide is a gaseous messenger that is the degration of heme.
血红素氧合酶是血红素降解的起始酶和限速酶，能代谢血红素形成一氧化碳、游离铁和胆红素。
Heme oxygenase which is heme-degradation enzyme and rate-limiting enzyme can catalyzes the degradation of heme to carbon monoxide, catalytic iron and bilirubin.
目的：研究血红素氧化酶1 (HO 1)基因在慢性肺心病大鼠支气管中的表达及分布情况。
Objective: To study the expression of heme oxygenase 1 (HO 1) in the bronchis of rats with chronic col pulmonale.
目的观察血红素氧化酶- 1 (HO - 1)在老年急性心肌梗死(ami)患者中的表达。
Objective To investigate the changes of heme oxygenase-1 (HO-1) in old patients with acute myocardial infarction (AMI).
前言：目的：探讨高血压时血红素氧合酶-一氧化碳系统的状态。
Objective: It is to explore the status of heme oxygenase carbon monoxide system in hypertension.
抗坏血酸在氯高铁血红素存在时，可以作为一个亲氧化剂而非一个抗氧化剂。
Ascorbic acid could function as a prooxidant, rather than an antioxidant, in the presence of hemin.
方法在比较使用联苯胺及其衍生物显现血手印的同时，实验了通过强氧化剂破坏血红素、沉淀珠蛋白，采用激活血痕、蛋白染色的方法，使血手印清晰显出。
Blood fingerprint was developed distinctly by using strong oxidation agent to destroy ferroheme, deposit pearl protein, also blood stain activation and protein decoration method were applied.
氢氧自由基淬灭剂显著地抑制抗坏血酸增强氯高铁血红素诱导红细胞溶血。由上述可得到如下结论：抗坏血酸增加氯高铁血红素诱导的红细胞氧化压力与H2O2有关；
Several HO. scavengers significantly inhibited hemo lysis induced by hemin and ascorbic acid. It was concluded that H2O2 was involved in the oxidative stress mediated by hemin and ascorbic acd.
其机制可能为一氧化碳抑制血红素蛋白氧感受器的功能。
The mechanism may be that CO binds to hemoprotein and inhibits hemoprotein oxygen sensor.
其机制可能为一氧化碳抑制血红素蛋白氧感受器的功能。
The mechanism may be that CO binds to hemoprotein and inhibits hemoprotein oxygen sensor.
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