结论吸入甲醛可加重哮喘大鼠气道阻力和气道炎症反应。
Conclusion Exposure to formaldehyde may exacerbate airway hyperresponsi-veness and inflammation in rat model of asthma.
以前研究表明吸入糖皮质激素减少炎症反应标记物(如c反应蛋白)和降低气道炎症反应。
Previous studies have shown inhaled corticosteroids reduce markers of inflammation such as C-reactive protein and reduce airway inflammation.
结论：PMN,IL - 6及IL - 8参与哮喘的气道炎症反应，而且哮喘缓解期患者的气道炎症反应持续存在。
Conclusions: PMN, IL-8 and IL-6 participate in development of airway inflammation in asthma. The airway inflammation still remains in stable asthmatics.
较为一般的情况是，室内烟雾中的微小颗粒物及其它污染物可使气道和双肺出现炎症，对免疫反应带来损害并且会降低血液携带氧气的能力。
More generally, small particulate matter and other pollutants in indoor smoke inflame the airways and lungs, impairing immune response and reducing the oxygen-carrying capacity of the blood.
背景与目的哮喘是慢性气道免疫性炎症，以气道淋巴细胞、嗜酸性粒细胞浸润、气道高反应性、粘液产生增加为特征。
Background and Objective Asthma is chronic airway immune inflammation characterized by airway lymphocyte infiltration and eosinophilia, airway hyperresponsiveness (AHR) and mucus hypersecretion.
慢性阻塞型肺疾病是一种气道性炎症性疾病，与肺对毒性颗粒或气体的异常炎症反应有关。
Chronic obstructive pulmonary disease (COPD) is an airway inflammation disease, And pulmonary abnormal inflammatory reaction to noxious gases or particles may be responsible to the disease.
结论痰诱导可行性好、不良反应发生率低，为慢性咳嗽的诊断、治疗提供了一项可靠的评价气道炎症的指标。
Induced sputum can provide a reliable data of airway inflammation for the patients with chronic nonproductive cough in clinical diagnosis and treatment.
在此项研究中，研究人员观察了具备哮喘加重病人主要特征的小鼠模型的气道炎症和对类固醇激素疗法的反应。
In this study, the researchers assessed airway inflammation and response to steroid treatment in a mouse model that replicates key features of asthmatic exacerbations observed in patients.
目的：研究致敏豚鼠不同强度的嗜酸细胞气道炎症对咳嗽反应的作用。
AIM: To investigate the effects of intensity of eosinophilic airway inflammation on cough response in guinea pigs sensitized and challenged with ovalbumin.
结论：缬沙坦可能是通过抑制AT 1受体抑制哮喘气道炎症和降低气道反应性。
Conclusion: Valsartan may inhibit airway inflammation and reduce airway responsiveness by suppressing AT1 receptors.
结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生，降低气道炎症和气道高反应性，这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
目的支气管哮喘(简称哮喘)是一种以气道慢性非特异性炎症和气道高反应为特征的常见肺部疾病。
Objective: Asthma is a pulmonary disease characterized by chronic inflammation of the airways and bronchial hyperresponsiveness.
阿司匹林可以影响炎症时涉及的（炎性）复合物和哮喘者存在的气道高反应性。
Aspirin can affect compounds involved in inflammation and the hyper-response of airways that is seen in asthma.
神经肽在气道高反应或气道炎症发生过程中的作用越来越引起重视。
Neuropeptide is causing more and more attentions for its role in airway hyperresponsiveness and in the occurrence of airway inflammation.
气道炎症细胞积聚、气道慢性炎症和气道高反应性是哮喘的三大重要特征。
Accumulation of airway inflammatory cells, chronic airway inflammation and airway hyperresponsiveness are three important features of asthma.
气道炎症细胞积聚、气道慢性炎症和气道高反应性是哮喘的三大重要特征。
Accumulation of airway inflammatory cells, chronic airway inflammation and airway hyperresponsiveness are three important features of asthma.
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