采取干预措施后，27家企业进行了治理，其中22家企业尘毒浓度达到国家卫生标准。
Intervention process made the dust and toxicant concentrations declined in 22 out of 27 factories to meet the demand of national hygienic standard.
结果蟾毒灵对ECV304细胞的生长有抑制作用，且呈浓度和时间的依赖性。
Result Bufalin had inhibition growth effect on ECV304 cells with different level, and had concentration and time dependence.
抗肿瘤药缓释制剂植入肿瘤组织、瘤周组织的间质中或肿瘤切除后的瘤床，达到局部高药物浓度，降低系统毒副反应。
The sustained-release anticancer drug implantation can achieve high local concentration with reducing systematic toxicity by implanting sustained-release drug into tumor tissue or interstitial tissue.
目的：探讨A型肉毒杆菌毒素(BTA)对豚鼠鼻黏膜腺细胞乙酰胆碱酯酶浓度的影响。
Objective: to investigate the effect of botulinum toxin type a (BTA) on the concentration of acetylcholinesterase in nasal glands of guinea pig.
结论：GLM提取物可提高了ADM对耐药细胞的细胞毒作用，增加了耐药细胞内药物浓度，且呈剂量依赖性。
CONCLUSION: GLM could enhance the cytotoxic effect of ADM on drug resistant cell and increase intracellular drug concentration in a concentration dependent fashion.
因此，5-FU血药浓度监测和毒副反应严重程度的预测具有重要的临床意义。
So monitoring serum concentration and predicting toxicity of 5-FU mean clinical sense.
试验结果表明，3个浓度的D型肉毒毒素对高原鼠兔的灭杀效果与0.1%的C型肉毒毒素相当。
The control effects of D type botulin at these 3 concentrations were equal to that of 0.1% C type botulin.
那是否需要对其进行常规的血药浓度监测？如何纠正其毒副反应。
Is it necessary to routinely test magnesium concentration and if toxicity occurs how should it be treated?
方法：细胞毒测定以MTT法；单药细胞毒测定以半倍浓度梯度设计；
Methods: Cytotoxicity of manumycin was determined by MTT assay, and cytotoxicity of the other drugs alone was determined using the half concentration gradient design.
方法指导癫痫患者规律用药，并做好服药期间药物毒副反应的观察，定期监测血象、肝功能及血药浓度等。
Methods Epileptic patients were instructed to regularize dose, taking. Dosing side, effects were observed, and routine blood tests, liver function and drug concentration of serum were monitored.
氟尿嘧啶植入剂可提高局部药物浓度，延长有效药物浓度时间，降低全身毒副反用。
Fluorouracil for implantation can elevate the local drug concentration, extend the time of effective drug concentration, reduce the general toxic and side-effect.
结果表明，TNF的直接细胞毒作用可致HUVEC转变成CEC的细胞形态，TNF浓度增加可能是病理情况下CEC增多的重要原因。
The results suggested that cytotoxicity of TNF can induce HUVEC to form CEC, which may be important in increasing CEC under certain pathological conditions.
结果表明，TNF的直接细胞毒作用可致HUVEC转变成CEC的细胞形态，TNF浓度增加可能是病理情况下CEC增多的重要原因。
The results suggested that cytotoxicity of TNF can induce HUVEC to form CEC, which may be important in increasing CEC under certain pathological conditions.
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