在一项研究中，我们观察了妊娠期肥胖患者，同时不仅母体全身存在某种程度的炎症反应，胎盘也有炎症反应。
Obviously in the study we have been conducting at least in the GDM obese women there are some level of inflammation not only systemic in the mother but also at the level of the placenta.
然而，另一个可能的解释包括反应产生的抗体从母体通过胎盘传递到胎儿。
However, another possible explanation involves the transfer of reactive antibodies from the mother through the placenta to the fetus.
胎盘是连接母体和婴儿的器官，为婴儿提供氧气和食物。
The placenta is the organ that unites the mother with the baby and provides air and food.
胎儿窘迫的主要相关因素有脐带因素、羊水因素、胎盘因素、母体因素等。
Fetal distress, umbilical cord, the main factors related factors, factors of amniotic fluid, placental factors, maternal factors.
而现实生活中由于母体、胎儿及胎盘因素导致的胎儿生长受限（FGR）实难避免。
While in our clinical work, we can often experienced such kind of newborns with fetal growth restriction (FGR) due to the factors of either mother, fetus or placenta.
结论HBV母婴传播可能还存在一种从胎盘母体面向胎儿的细胞转移的方式；
Conclusion It may become true by the way transferred between the cells from the mother side to the fetus side in the placenta about maternal fetal transmission of HBV.
相比之下，胎儿并没有非常经典的炎症反应。我们知道，母体循环中的细胞因子并不能透过胎盘，因此不能到达胎儿。
By contrast the fetus is not in a very classic inflammatory state and we know that the cytokines which are circulating in the mother cannot cross the placenta so they do not reach the fetus.
在妊娠期较晚时的测试性能的提高可能与随着胎儿和胎盘的发育母体血液中无细胞胎儿dna浓度的增加有关。
The improved performance with later gestation is likely attributable to the increased concentration of cell-free fetal DNA within maternal blood as the fetus and placenta develop.
在妊娠期较晚时的测试性能的提高可能与随着胎儿和胎盘的发育母体血液中无细胞胎儿dna浓度的增加有关。
The improved performance with later gestation is likely attributable to the increased concentration of cell-free fetal DNA within maternal blood as the fetus and placenta develop.
应用推荐